Associate Professor of Medicine, Boston University
Endocrinology
Dr. Alan P. Farwell, MD
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Alan P. Farwell practices Endocrinology. He attended University of Massachusetts Medical School. University of Massachusetts Medical School is ranked 49/12 in Research/PrimaryCare. He received 2 awards: "Boston Magazine" and "Super Doctor". Alan Farwell is a published doctor. He has 39 papers published. The most current publication "Thyroid hormone-regulated actin polymerization in brain." Alan Farwell is registered with Medicare and accepts Medicare payments.
Publications
- NonThyroidal illness syndrome.
- Thyrotoxicosis with post-treatment hypoThyroidism in a patient with acute suppurative Thyroiditis due to porphyromonas.
- E. Chester Ridgway Delivers the First Lewis E. Braverman Distinguished Award Lectureship at the American Thyroid Association's 2011 Annual Meeting.
- Thyroid hormone therapy is not indicated in the majority of patients with the sick euThyroid syndrome.
- Unsatisfactory consequences from fine-needle aspiration biopsy of Thyroid nodules.
- Dynamic nongenomic actions of Thyroid hormone in the developing rat brain.
- Thyroiditis.
- Modulation of myocardial L-triiodothyronine receptors in normal, hypoThyroid, and hyperThyroid rats.
- Total suppression of cortisol excretion by ketoconazole in the therapy of the ectopic adrenocorticotropic hormone Syndrome.
- Selenium-regulated translation control of heterologous gene expression: normal function of selenoCysteine-substituted gene products.
- Evidence that Type II 5'-deiodinase is not a selenoprotein.
- Thyroidectomy for amiodarone-induced thyrotoxicosis.
- Tissue-engineered spinal cord.
- Inflammatory Thyroid disorders.
- Nuclear Thyroid hormone receptor in the rat uterus.
- Localization of Type I iodothyronine 5'-deiodinase to the basolateral plasma membrane in renal cortical epithelial cells.
- The Thyroid gland is a major source of circulating T3 in the rat.
- The actin cytoskeleton mediates the hormonally regulated translocation of Type II iodothyronine 5'-deiodinase in astrocytes.
- The mammalian homolog of the frog Type II selenodeiodinase does not encode a functional enzyme in the rat.
- Identification of a 27-kDa protein with the properties of Type II iodothyronine 5'-deiodinase in dibutyryl cyclic AMP-stimulated glial cells.
- Degradation and recycling of the substrate-binding subunit of Type II iodothyronine 5'-deiodinase in astrocytes.
- Structural requirements of iodothyronines for the rapid inactivation and internalization of Type II iodothyronine 5'-deiodinase in glial cells.
- Real-time visualization of processive myosin 5a-mediated vesicle movement in living astrocytes.
- Cloning, expression, and functional characterization of the substrate binding subunit of rat Type II iodothyronine 5'-deiodinase.
- Differential expression of Thyroid hormone receptor isoforms in neurons and astroglial cells.
- Thyroxine targets different pathways of internalization of Type II iodothyronine 5'-deiodinase in astrocytes.
- Effects of selenium deficiency on Thyroid hormone economy in rats.
- Thyroid hormone-dependent redistribution of the 55-kilodalton monomer of protein disulfide isomerase in cultured glial cells.
- Catalytic activity of Type II iodothyronine 5'-deiodinase polypeptide is dependent upon a cyclic AMP activation factor.
- Thyroid hormone regulates the extracellular organization of laminin on astrocytes.
- Thyroxine-dependent regulation of integrin-laminin interactions in astrocytes.
- Myosin V plays an essential role in the Thyroid hormone-dependent endocytosis of type II iodothyronine 5'-deiodinase.
- Dissociation of actin polymerization and enzyme inactivation in the hormonal regulation of Type II iodothyronine 5'-deiodinase activity in astrocytes.
- Selenium deficiency and type II 5'-deiodinase regulation in the euThyroid and hypoThyroid rat: evidence of a direct effect of thyroxine.
- Thyroid hormone-regulated actin polymerization in brain.
- Thyroid hormone regulates the expression of laminin in the developing rat cerebellum.
- Regulation of cerebellar neuronal migration and neurite outgrowth by thyroxine and 3,3'',5''-triiodothyronine.
- Effect of methyl iodide on deiodinase activity.
Schools
University of Massachusetts Worcester Campus
Mc Hospital Of Vt
University Mass Med Center
Conditions Treated
- Abnormal Thyroid
- Adrenal Incidentaloma
- Adrenal Insufficiency
- Calcium Metabolism Disorders
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Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- Boston Magazine
- Super Doctor
Education
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University of Massachusetts -
University of Vermont - Fletcher Allen Health Care -
University of Massachusetts Medical School
Hospital
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Boston Medical Center
Drug Facts
| NPI NUMBER | 1699758151 | |
| NPPES Provider LastName | FARWELL | |
| NPPES Provider FirstName | ALAN | |
| NPPES Provider ZIPCode | 021182309 | |
| NPPES Provider State | MA | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 686.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1699758151 |
| Last Name Of The Provider | FARWELL |
| First Name Of The Provider | ALAN |
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