Assistant Professor, Pediatrics, University of Utah School of Medicine
Pediatrics
Dr. Andrew G. Smith is an expert in the field of Pediatrics. He went to Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is one of the top medical schools in the US. Johns Hopkins University School of Medicine is ranked 3 in research and 29 in primary care medicine He has 1 awards "Fellow (FAAP)". Andrew Smith is a published physician. He published 31 publications, including: 'Pediatric Palliative Care and Inpatient Hospital Costs: A Longitudinal Cohort Study.'
Publications
- Pediatric Palliative Care and Inpatient Hospital Costs: A Longitudinal Cohort Study.
- Early Failure of N-Methyl-D-aspartate Receptors and Deficient Spine Formation Induced by Reduction of Regulatory Heme in Neurons.
- Mevastatin accelerates loss of synaptic proteins and neurite degeneration in aging cortical neurons in a heme-independent manner.
- Complex gene-chemical interactions: hepatic uroporphyria as a paradigm.
- Patients with both basal and squamous cell carcinomas are at a lower risk of further basal cell carcinomas than patients with only a basal cell carcinoma.
- The value of skin biopsy in inflammatory dermatoses.
- B2 SINE retrotransposon causes polymorphic expression of mouse 5-aminolevulinic acid synthase 1 gene.
- Single-molecule PCR analysis of germ line mutation induction by anticancer drugs in mice.
- Polychlorinated dibenzo-p-dioxins and the human immune system: 4 studies on two Spanish families with increased body burdens of highly chlorinated PCDDs.
- Essential role of the AH receptor in the dysfunction of heme metabolism induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
- Seven-year prospective study of nonmelanoma skin cancer incidence in U.K. renal transplant recipients.
- Neurite degeneration induced by heme deficiency mediated via inhibition of NMDA receptor-dependent extracellular signal-regulated kinase 1/2 activation.
- Hexachlorobenzene stimulates uroporphyria in low affinity AHR mice without increasing CYP1A2.
- Dihydroxy-, hydroxyspirolactone-, and dihydroxyspirolactone-urochlorins induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in the liver of mice.
- Sebaceous hyperplasia and skin cancer in patients undergoing renal transplant.
- Multiple polymorphic loci determine basal hepatic and splenic iron status in mice.
- Heme deficiency is associated with senescence and causes suppression of N-methyl-D-aspartate receptor subunits expression in primary cortical neurons.
- Uroporphyria and hepatic carcinogenesis induced by polychlorinated biphenyls-iron interaction: absence in the Cyp1a2(-/-) knockout mouse.
- Polymorphism in the nuclear excision repair gene ERCC2/XPD: association between an exon 6-exon 10 haplotype and susceptibility to cutaneous basal cell carcinoma.
- Hepatic gene expression in protoporphyic Fech mice is associated with cholestatic injury but not a marked depletion of the heme regulatory pool.
- Associations between ultraviolet radiation, basal cell carcinoma site and histology, host characteristics, and rate of development of further tumors.
- Cyp1a1(-/-) male mice: protection against high-dose TCDD-induced lethality and wasting syndrome, and resistance to intrahepatocyte lipid accumulation and uroporphyria.
- PTCH polymorphism is associated with the rate of increase in basal cell carcinoma numbers during follow-up: preliminary data on the influence of an exon 12-exon 23 hap...
- Factors associated with nonmelanoma skin cancer following renal transplantation in Queensland, Australia.
- Gene expression profiles associated with inflammation, fibrosis, and cholestasis in mouse liver after griseofulvin.
- Intrinsic hepatic phenotype associated with the Cyp1a2 gene as shown by cDNA expression microarray analysis of the knockout mouse.
- Hepatobiliary damage and changes in hepatic gene expression caused by the antitumor drug ecteinascidin-743 (ET-743) in the female rat.
- A model for nurse-led skin cancer surveillance following renal transplantation.
- Non-ahr gene susceptibility Loci for porphyria and liver injury induced by the interaction of 'dioxin' with iron overload in mice.
- Changes to Pediatric Clerkships' Nighttime Structure After Introduction of the 2011 ACGME Resident Duty Hour Standards.
Schools
Johns Hopkins University School of Medicine
Doctors Specialties
Accepted Insurances
Awards
Education
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University of Utah
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Johns Hopkins University School of Medicine
Hospital
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University of Utah Health Care - Hospital and Clinics
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