Physician
General Surgery
Dr. Brent R. Weil, MD
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Brent Weil is an expert in the field of General Surgery. He researched medicine at Indiana University School of Medicine. Indiana University School of Medicine is ranked 45/47 in Research/PrimaryCare. He over the years received 1 awards: "Fellow (FACS)". Brent Weil is a published doctor and has 54 publications published. The lastest was: 'Prospective determination of percutaneous endoscopic gastrostomy complication rates in children: Still a safe procedure.'
Publications
- Prospective determination of percutaneous endoscopic gastrostomy complication rates in children: Still a safe procedure.
- The jury is still out: changes in gastroschisis management over the last decade are associated with both benefits and shortcomings.
- Pretreating mesenchymal stem cells with interleukin-1? and transforming growth factor-? synergistically increases vascular endothelial growth factor production and imp...
- Predictive value of response to steroid therapy on response to splenectomy in children with immune thrombocytopenic purpura.
- Transforming growth factor-α enhances stem cell-mediated postischemic myocardial protection.
- Transforming growth factor-alpha does not protect myocardium during acute ischemia/reperfusion.
- Pretreatment with intracoronary mimosine improves postischemic myocardial functional recovery.
- Systemic pretreatment with dimethyloxalylglycine increases myocardial HIF-1? and VEGF production and improves functional recovery after acute ischemia/reperfusion.
- Interleukin-10 protects the ischemic heart from reperfusion injury via the STAT3 pathway.
- Intravenous Infusion of Mesenchymal Stem Cells is Associated with Improved Myocardial Function During Endotoxemia.
- Intracoronary Mesenchymal Stem Cells Promote Postischemic Myocardial Functional Recovery, Decrease Inflammation, and Reduce Apoptosis via a Signal Transducer and Activ...
- IL-6 and TGF-α costimulate mesenchymal stem cell vascular endothelial growth factor production by ERK-, JNK-, and PI3K-mediated mechanisms.
- The immunomodulatory properties of mesenchymal stem cells: implications for surgical disease.
- FEMALE STEM CELLS ARE SUPERIOR TO MALES IN PRESERVING MYOCARDIAL FUNCTION FOLLOWING ENDOTOXEMIA.
- Exogenous high-mobility group box 1 improves myocardial recovery after acute global ischemia/reperfusion injury.
- Optimizing stem cell function for the treatment of ischemic heart disease.
- Toll-like receptor signaling pathways and the evidence linking toll-like receptor signaling to cardiac ischemia/reperfusion injury.
- The phosphoinositide-3 kinase survival signaling mechanism in sepsis.
- Ablation of TNF-alpha receptors influences mesenchymal stem cell-mediated cardiac protection against ischemia.
- Signaling via GPR30 protects the myocardium from ischemia/reperfusion injury.
- Animal models of myocardial and vascular injury.
- Pericardial effusion and cardiac tamponade associated with central venous catheters in children: an uncommon but serious and treatable condition.
- Mesenchymal stem cells attenuate myocardial functional depression and reduce systemic and myocardial inflammation during endotoxemia.
- Postinfarct intramyocardial injection of mesenchymal stem cells pretreated with TGF-alpha improves acute myocardial function.
- TNF receptor 2, not TNF receptor 1, enhances mesenchymal stem cell-mediated cardiac protection following acute ischemia.
- Toll-like receptor 2 mediates mesenchymal stem cell-associated myocardial recovery and VEGF production following acute ischemia-reperfusion injury.
- Gender dimorphisms in progenitor and stem cell function in cardiovascular disease.
- Preconditioning mesenchymal stem cells with transforming growth factor-alpha improves mesenchymal stem cell-mediated cardioprotection.
- TLR4 inhibits mesenchymal stem cell (MSC) STAT3 activation and thereby exerts deleterious effects on MSC-mediated cardioprotection.
- MEK, p38, and PI-3K mediate cross talk between EGFR and TNFR in enhancing hepatocyte growth factor production from human mesenchymal stem cells.
- What's new in Shock, October 2009?
- MEK mediates the novel cross talk between TNFR2 and TGF-EGFR in enhancing vascular endothelial growth factor (VEGF) secretion from human mesenchymal stem cells.
- Mesenchymal stem cells enhance the viability and proliferation of human fetal intestinal epithelial cells following hypoxic injury via paracrine mechanisms.
- Proinflammatory stem cell signaling in cardiac ischemia.
- Signal transducer and activator of transcription 3-stimulated hypoxia inducible factor-1alpha mediates estrogen receptor-alpha-induced mesenchymal stem cell vascular e...
- Stem cells in sepsis.
- High glucose concentration in cell culture medium does not acutely affect human mesenchymal stem cell growth factor production or proliferation.
- Proestrus female rats are more resistant to right ventricular pressure overload.
- Spontaneous duodenal hematoma: a rare cause of upper gastrointestinal tract obstruction.
- The effects of estrogen on pulmonary artery vasoreactivity and hypoxic pulmonary vasoconstriction: potential new clinical implications for an old hormone.
- Bleeding risk for surgical dialysis procedures in children with hemolytic uremic syndrome.
- Surgical treatment of atrial fibrillation: the time is now.
- Cell-based therapy for ischemic heart disease: a clinical update.
- Exogenous estrogen rapidly attenuates pulmonary artery vasoreactivity and acute hypoxic pulmonary vasoconstriction.
- Proinflammatory cytokine effects on mesenchymal stem cell therapy for the ischemic heart.
- Both endogenous and exogenous testosterone decrease myocardial STAT3 activation and SOCS3 expression after acute ischemia and reperfusion.
- Acute postischemic treatment with estrogen receptor-alpha agonist or estrogen receptor-beta agonist improves myocardial recovery.
- Deleterious effects of endogenous and exogenous testosterone on mesenchymal stem cell VEGF production.
- Nitric oxide suppresses the secretion of vascular endothelial growth factor and hepatocyte growth factor from human mesenchymal stem cells.
- Role of tumor necrosis factor receptor 1 in sex differences of stem cell mediated cardioprotection.
- Estrogen receptor beta mediates increased activation of PI3K/Akt signaling and improved myocardial function in female hearts following acute ischemia.
- Prehypertension and endothelial progenitor cell function.
- Bcl-2 protein in normal, hyperplastic and neoplastic breast tissues. A metabolite of the putative stem-cell subpopulation of the mammary gland.
Schools
Ohio State University College of Medicine
Doctors Specialties
- Pediatric Surgery
- General Surgery
Accepted Insurances
Awards
- Fellow (FACS)
Education
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Boston Children's Hospital -
Indiana University School of Medicine -
The Ohio State University College of Medicine
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