Associate Professor, Internal Medicine, University of South Alabama College of Medicine
Pulmonology
Dr. Brian W. Fouty, MD
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Brian Fouty works in the field of Pulmonology. He studied medicine at University of Washington School of Medicine. Dr. Brian W. Fouty is also published. He has 34 publications published. The lastest publication: "The pulmonary circulation of homozygous or heterozygous eNOS-null mice is hyperresponsive to mild hypoxia.'
Publications
- Overlap of Doxycycline Fluorescence with that of the Redox-Sensitive Intracellular Reporter roGFP.
- Minimal piggyBac vectors for chromatin integration.
- Functional and genomic changes induced by alveolar transmigration in human neutrophils.
- Nitric oxide augments fetal Pulmonary artery endothelial cell angiogenesis in vitro.
- Suppression of type II bone morphogenic protein receptor in vascular smooth muscle induces Pulmonary arterial hypertension in transgenic mice.
- Recombinant human activated protein C reduces human endotoxin-induced Pulmonary inflammation via inhibition of neutrophil chemotaxis.
- Mevastatin can cause G1 arrest and induce apoptosis in Pulmonary artery smooth muscle cells through a p27Kip1-independent pathway.
- Serum can overcome contact inhibition in confluent human Pulmonary artery smooth muscle cells.
- Heterogeneous activation of p19Arf in Pulmonary artery smooth muscle cells.
- Mifepristone increases gamma-retroviral infection efficiency by enhancing the integration of virus into the genome of infected cells.
- Dexamethasone and mifepristone increase retroviral infectivity through different mechanisms.
- Diabetes and the Pulmonary circulation.
- Pulmonary microvascular endothelial cells form a tighter monolayer when grown in Chronic hypoxia.
- Proproliferative phenotype of Pulmonary microvascular endothelial cells.
- Dexamethasone can stimulate G1-S phase transition in human airway fibroblasts in Asthma.
- Pulmonary hypertension in transgenic mice expressing a dominant-negative BMPRII gene in smooth muscle.
- Low-voltage-activated (T-type) calcium channels control proliferation of human Pulmonary artery myocytes.
- Tumor necrosis factor-related apoptosis-inducing ligand can induce apoptosis in subsets of premalignant cells.
- Pulmonary vascular gene transfer: Prospects for successful therapy of Pulmonary hypertension.
- Riboflavin to treat nucleoside analogue-induced lactic Acidosis.
- Protein kinase G is not essential to NO-cGMP modulation of basal tone in rat Pulmonary circulation.
- Cytosolic Ca2+ and adenylyl cyclase responses in phenotypically distinct Pulmonary endothelial cells.
- Inhibition of cyclic 3'-5'-guanosine monophosphate-specific phosphodiesterase selectively vasodilates the pulmonary circulation in Chronically hypoxic rats.
- Reduced PO2 alters the behavior of Fura-2 and Indo-1 in bovine Pulmonary artery endothelial cells.
- Enhanced ET(A)-receptor-mediated inhibition of K(v) channels in hypoxic hypertensive rat Pulmonary artery myocytes.
- The Pulmonary circulation of homozygous or heterozygous eNOS-null mice is hyperresponsive to mild hypoxia.
- Role of neutrophils in generalized reperfusion injury associated with resuscitation from shock.
- Dilatation of bronchial stenoses due to Sarcoidosis using a flexible fiberoptic bronchoscope.
- Hypoxemia explained 36 years later.
- p27(Kip1) is important in modulating Pulmonary artery smooth muscle cell proliferation.
- Upregulation of nitric oxide synthase in mice with severe hypoxia-induced Pulmonary hypertension.
- Relative contributions of endothelial, inducible, and neuronal NOS to tone in the murine Pulmonary circulation.
- Thrombospondin-1, endothelium and systemic vascular tone.
Schools
University of Washington
Procedures Preformed
- Bronchoscopy
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Atelectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Education
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University of Arizona -
University of Washington School of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1740234327 |
| Last Name Of The Provider | FOUTY |
| First Name Of The Provider | BRIAN |
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