Professor, Medicine, Medical College of Georgia at Georgia Health Sciences University
Endocrinology
Dr. Carlos M. Isales, MD
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Carlos Isales works in the field of Endocrinology. He researched medicine at Yale University School of Medicine. Yale University School of Medicine is one of the top medical schools in the US. Yale University School of Medicine has a rank of 7 in research and a 57 in primary care He over the years received 1 awards: "Regional Top Doctor". Carlos M. Isales has written publications. He has 101 publications published with PubMed. The lastest publication is: 'Fenfluramine potentiates canine pulmonary vasoreactivity to endothelin-1.' Carlos Isales accepts Medicare.
Publications
- Deregulation of arginase induces bone complications in high-fat/high-sucrose diet diabetic mouse model.
- Insulin resistance negatively influences the muscle-dependent IGF-I-bone mass relationship in pre-menarcheal girls.
- Zinc Supplementation Increases Procollagen Type 1 Amino-Terminal Propeptide in Premenarcheal Girls: A Randomized Controlled Trial.
- Caloric restriction and the adipokine leptin alter the SDF-1 signaling axis in bone marrow and in bone marrow derived mesenchymal stem cells.
- Mesenchymal stem cell expression of stromal cell-derived factor-1β augments bone formation in a model of local regenerative therapy.
- Impact of targeted PPARγ disruption on bone remodeling.
- MicroRNAs-141 and 200a regulate the SVCT2 transporter in bone marrow stromal cells.
- Oxidation of the aromatic amino acids tryptophan and tyrosine disrupts their anabolic effects on bone marrow mesenchymal stem cells.
- Impact of dietary aromatic amino acids on osteoclastic activity.
- Total Body Irradiation Is Permissive For Mesenchymal Stem Cell-Mediated New Bone Formation Following Local Transplantation.
- Role of Glucocorticoid-Induced Leucine Zipper (GILZ) in Bone Acquisition.
- Low-Dose Bone Morphogenetic Protein-2 / Stromal Cell-Derived Factor-1β Co-Therapy Induces Bone Regeneration in Critical-Size Rat Calvarial Defects.
- GRowing an Epidermal Tumor.
- Knockdown of SVCT2 impairs in-vitro cell attachment, migration and wound healing in bone marrow stromal cells.
- A myostatin inhibitor (propeptide-Fc) increases muscle mass and muscle fiber size in aged mice but does not increase bone density or bone strength.
- Regulation of vitamin C transporter in the type 1 diabetic mouse bone and bone marrow.
- Pine Oil Effects on Chemical and Thermal Injury in Mice and Cultured Mouse Dorsal Root Ganglion Neurons.
- Stromal Cell-Derived Factor-1? Mediates Cell Survival through Enhancing Autophagy in Bone Marrow-Derived Mesenchymal Stem Cells.
- Sodium-coupled vitamin C transporter (SVCT2): expression, function, and regulation in intervertebral disc cells.
- Effects of the activin A-myostatin-follistatin system on aging bone and muscle progenitor cells.
- Sodium-dependent vitamin C transporter SVCT2: Expression and function in bone marrow stromal cells and in osteogenesis.
- Effect of KCNJ5 Mutations on Gene Expression in Aldosterone-Producing Adenomas and Adrenocortical Cells.
- Stromal Cell-Derived Factor-1β Potentiates Bone Morphogenetic Protein-2-Stimulated Osteoinduction of Genetically Engineered Bone Marrow-Derived Mesenchymal Stem Cells ...
- Effect of whole-body vibration on bone properties in aging mice.
- The adipokine leptin increases skeletal muscle mass and significantly alters skeletal muscle miRNA expression profile in aged mice.
- ACTH is a novel regulator of bone mass.
- Restoration of regenerative osteoblastogenesis in aged mice: modulation of TNF.
- Age-related changes in the osteogenic differentiation potential of mouse bone marrow stromal cells.
- Caloric restriction decreases cortical bone mass but spares trabecular bone in the mouse skeleton: implications for the regulation of bone mass by body weight.
- Impact of glucose-dependent insulinotropic peptide on age-induced bone loss.
- Future developments in therapy.
- Role of the oral and maxillofacial surgeon in the diagnosis and treatment of patients with osteoporosis.
- Pituitary glycoprotein hormone receptors in non-endocrine organs.
- Glucose-dependent insulinotropic peptide-overexpressing transgenic mice have increased bone mass.
- Effects of glucose-dependent insulinotropic peptide on osteoclast function.
- Characterization and phospholipase D mediation of the angiotensin II priming response in adrenal glomerulosa cells.
- Energy Balance, Myostatin, and GILZ: Factors Regulating Adipocyte Differentiation in Belly and Bone.
- Effects of glucose-dependent insulinotropic peptide on behavior.
- Age-related loss of muscle mass and bone strength in mice is associated with a decline in physical activity and serum leptin.
- Disordered osteoclast formation in RAGE-deficient mouse establishes an essential role for RAGE in diabetes related bone loss.
- Glucose-dependent insulinotropic polypeptide receptor knockout mice have altered bone turnover.
- Muscle-bone interactions in dystrophin-deficient and myostatin-deficient mice.
- Multiple melanocortin receptors are expressed in bone cells.
- Carboxy-terminal PTH fragments stimulate [3H]thymidine incorporation in vascular endothelial cells.
- A patient with concurrent primary hyperaldosteronism and adrenal insufficiency.
- Glucose-dependent insulinotropic peptide: differential effects on hepatic artery vs. portal vein endothelial cells.
- Tension-induced reduction in connexin 43 expression in cranial sutures is linked to transcriptional regulation by TBX2.
- High glucose augments the angiotensin II-induced activation of JAK2 in vascular smooth muscle cells via the polyol pathway.
- Glucose-dependent insulinotropic peptide stimulates thymidine incorporation in endothelial cells: role of endothelin-1.
- Negative transcriptional regulation of connexin 43 by Tbx2 in rat immature coronal sutures and ROS 17/2.8 cells in culture.
- Glucose-dependent insulinotropic peptide stimulates proliferation and TGF-beta release from MG-63 cells.
- Parathyroid hormone effects on signaling pathways in endothelial cells vary with peptide concentration.
- Stem Cell-Derived Exosomes: A Potential Alternative Therapeutic Agent in Orthopaedics.
- Aromatic amino Acid activation of signaling pathways in bone marrow mesenchymal stem cells depends on oxygen tension.
- Absence of Functional Leptin Receptor Isoforms in the POUND (Lepr(db/lb)) Mouse Is Associated with Muscle Atrophy and Altered Myoblast Proliferation and Differentiation.
- ANP-(7-23) stimulates a DHP-sensitive Ca2+ conductance and reduces cellular cAMP via a cGMP-independent mechanism.
- microRNA deficiency in pancreatic islet cells exacerbates streptozotocin-induced murine autoimmune diabetes.
- Skeletal receptors for steroid-family regulating glycoprotein hormones: A multilevel, integrated physiological control system.
- 25-hydroxyvitamin D, insulin-like growth factor-I, and bone mineral accrual during growth.
- A ;C' for the Pancreatic ABCs.
- CART deficiency increases body weight but does not alter bone strength.
- Targeted disruption of the Lasp-1 gene is linked to increases in histamine-stimulated gastric HCl secretion.
- Spontaneous bone loss in RIP-iNOS transgenic mouse: a mouse model for diabetes-mediated osteopenia/osteoporosis.
- ACTH protects against glucocorticoid-induced osteonecrosis of bone.
- Nutritional hormones and the entero-osseous axis.
- Resistance to body fat gain in ''double-muscled'' mice fed a high-fat diet.
- Loss of myostatin (GDF8) function increases osteogenic differentiation of bone marrow-derived mesenchymal stem cells but the osteogenic effect is ablated with unloading.
- Tbx2 represses expression of Connexin43 in osteoblastic-like cells.
- The LTR enhancer of ERV-9 human endogenous retrovirus is active in oocytes and progenitor cells in transgenic zebrafish and humans.
- Tauroursodeoxycholic acid stimulates hepatocellular exocytosis and mobilizes extracellular Ca++ mechanisms defective in cholestasis.
- Primary ovarian lymphoma manifesting with severe hypercalcemia.
- A midregion parathyroid hormone-related peptide mobilizes cytosolic calcium and stimulates formation of inositol trisphosphate in a squamous carcinoma cell line.
- Tauroursodeoxycholic acid activates protein kinase C in isolated rat hepatocytes.
- Diacylglycerol production, Ca2+ influx, and protein kinase C activation in sustained cellular responses.
- Endothelin-1 induces cholestasis which is mediated by an increase in portal pressure.
- Angiotensin II priming of aldosterone secretion with agents that enhance Ca(2+) influx.
- Microarray analysis of Tbx2-directed gene expression: a possible role in osteogenesis.
- Glucose-dependent insulinotropic peptide is an integrative hormone with osteotropic effects.
- Functional analysis of glucose-dependent insulinotropic polypeptide fusion proteins.
- Differential effects of agonists of aldosterone secretion on steroidogenic acute regulatory phosphorylation.
- Glucose-dependent insulinotropic peptide signaling pathways in endothelial cells.
- Functional parathyroid hormone receptors are present in an umbilical vein endothelial cell line.
- Osteoblast-derived cells express functional glucose-dependent insulinotropic peptide receptors.
- Acetoacetate and beta-hydroxybutyrate differentially regulate endothelin-1 and vascular endothelial growth factor in mouse brain microvascular endothelial cells.
- Fenfluramine potentiates canine pulmonary vasoreactivity to endothelin-1.
- Overexpression of protein kinase C alpha and beta1 has distinct effects on bovine aortic endothelial cell growth.
- pH-dependent fluoride transport in intestinal brush border membrane vesicles.
- Atrial natriuretic peptide differentially modulates T- and L-type calcium channels.
- Effects of Ca2+ agonists on cytosolic Ca2+ in isolated hepatocytes and on bile secretion in the isolated perfused rat liver.
- Ca2+ channels and aldosterone secretion: modulation by K+ and atrial natriuretic peptide.
- Angiotensin-II-induced changes in diacylglycerol levels and their potential role in modulating the steroidogenic response.
- A potential role for phospholipase-D in the angiotensin-II-induced stimulation of aldosterone secretion from bovine adrenal glomerulosa cells.
- Modulation of Ca2+ channels by atrial natriuretic peptide in the bovine adrenal glomerulosa cell.
- Parathyroid hormone modulates angiotensin II-induced aldosterone secretion from the adrenal glomerulosa cell.
- Signal transduction mechanisms involved in carbachol-induced aldosterone secretion from bovine adrenal glomerulosa cells.
- Role of calcium in angiotensin II-mediated aldosterone secretion.
- Effect of ANP on sustained aldosterone secretion stimulated by angiotensin II.
- Short term memory in the calcium messenger system. Evidence for a sustained activation of protein kinase C in adrenal glomerulosa cells.
- Atrial natriuretic peptide inhibits the stimulation of aldosterone secretion but not the transient increase in intracellular free calcium concentration induced by angi...
- Effect of short-term somatostatin and long-term triiodothyronine administration in a child with nontumorous inappropriate thyrotropin secretion.
Schools
Univ Of Pr Sch Of Med
San Juan Va Hospital
Yale New Haven Hospital
Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- Regional Top Doctor
Education
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Yale University School of Medicine -
University of Puerto Rico School of Medicine
Hospital
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Georgia Regents Medical Center
Drug Facts
| NPI NUMBER | 1740390061 | |
| NPPES Provider LastName | ISALES | |
| NPPES Provider FirstName | CARLOS | |
| NPPES Provider ZIPCode | 309120004 | |
| NPPES Provider State | GA | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 1130.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1740390061 |
| Last Name Of The Provider | ISALES |
| First Name Of The Provider | CARLOS |
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