Professor, Pathology, University of Alabama School of Medicine
Pathology
Dr. Casey T. Weaver, MD
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Doctor Casey T. Weaver, MD practices Pathology. He graduated from University of Florida College of Medicine. University of Florida College of Medicine has a rank of 43 in research and a 78 in primary care Casey Weaver is also published. He has 99 publications published. The lastest publication: "Experimental models to study molecular mechanisms underlying intestinal inflammation.' Casey T. Weaver, MD accepts Medicare payments and is listed with Medicare.gov.
Publications
- Daughter's Tolerance of Mom Matters in Mate Choice.
- IL-1 signaling modulates activation of STAT transcription factors to antagonize retinoic acid signaling and control the TH17 cell-iTreg cell balance.
- Development and Survival of Th17 Cells within the Intestines: The Influence of Microbiome- and Diet-Derived Signals.
- The Transcription Factors T-bet and Runx Are Required for the Ontogeny of Pathogenic Interferon-γ-Producing T Helper 17 Cells.
- Deletion of a conserved cis-element in the Ifng locus highlights the role of acute histone acetylation in modulating inducible gene transcription.
- IL-2 coordinates IL-2-producing and regulatory T cell interplay.
- Peritoneal cavity regulatory B cells (B10 cells) modulate IFN-γ+CD4+ T cell numbers during colitis development in mice.
- Notch simultaneously orchestrates multiple helper T cell programs independently of cytokine signals.
- IL-22-producing neutrophils contribute to antimicrobial defense and restitution of colonic epithelial integrity during colitis.
- Dwelling on T cell fate decisions.
- Linking Vitamin D Deficiency to Inflammatory Bowel Disease.
- The Th17 family: flexibility follows function.
- Allogeneic Th1 Cells Home to Host Bone Marrow and Spleen and Mediate IFN?-Dependent Aplasia.
- Retinoic Acid Hypersensitivity Promotes Peripheral Tolerance in Recent Thymic Emigrants.
- Role of TLR2-dependent IL-10 production in the inhibition of the initial IFN-? T cell response to Porphyromonas gingivalis.
- Th22 Cells Are an Important Source of IL-22 for Host Protection against Enteropathogenic Bacteria.
- The Th17 pathway and inflammatory diseases of the intestines, lungs, and skin.
- The β-Glucan Receptor Dectin-1 Promotes Lung Immunopathology during Fungal Allergy via IL-22.
- Regulatory b10 cells differentiate into antibody-secreting cells after transient IL-10 production in vivo.
- Th17 cells mediate clade-specific, serotype-independent mucosal immunity.
- Natural killer cell activation enhances immune pathology and promotes chronic infection by limiting CD8+ T-cell immunity.
- B cell-derived IL-10 does not regulate spontaneous systemic autoimmunity in MRL.Fas(lpr) mice.
- Developmental regulation of Th17-cell capacity in human neonates.
- Dectin-1 dependent IL-22 contributes to early innate lung defense against Aspergillus fumigatus.
- Neutrophils produce IL-17A in a Dectin-1 and IL-23 dependent manner during invasive fungal infection.
- Interleukin-12 converts Foxp3+ regulatory T cells to interferon-γ-producing Foxp3+ T cells that inhibit colitis.
- Lineage-specific effects of 1,25-dihydroxyvitamin D(3) on the development of effector CD4 T cells.
- Pronounced virus-dependent activation drives exhaustion but sustains IFN-γ transcript levels.
- Modular utilization of distal cis-regulatory elements controls Ifng gene expression in T cells activated by distinct stimuli.
- Epigenetic instability of cytokine and transcription factor gene loci underlies plasticity of the T helper 17 cell lineage.
- Autoimmunity: increasing suspects in the CD4+ T cell lineup.
- Intestinal effector T cells in health and disease.
- IL-9 as a mediator of Th17-driven inflammatory disease.
- TGF-beta promotes Th17 cell development through inhibition of SOCS3.
- Contrasting roles for all-trans retinoic acid in TGF-beta-mediated induction of Foxp3 and Il10 genes in developing regulatory T cells.
- Late developmental plasticity in the T helper 17 lineage.
- Emergence of the Th17 pathway and its role in host defense.
- Regulatory T cells expressing interleukin 10 develop from Foxp3+ and Foxp3- precursor cells in the absence of interleukin 10.
- Monoclonal anti-interleukin 23 reverses active colitis in a T cell-mediated model in mice.
- IL-17 family cytokines and the expanding diversity of effector T cell lineages.
- A distal conserved sequence element controls Ifng gene expression by T cells and NK cells.
- Th17: an effector CD4 T cell lineage with regulatory T cell ties.
- Antigen and lipopolysaccharide play synergistic roles in the effector phase of airway inflammation in mice.
- Alterations of T lymphocytes in inflammatory bowel diseases.
- Both Th1 and Th2 cells require P-selectin glycoprotein ligand-1 for optimal rolling on inflamed endothelium.
- P-selectin can support both Th1 and Th2 lymphocyte rolling in the intestinal microvasculature.
- Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages.
- Generation of antigen-specific, Foxp3-expressing CD4+ regulatory T cells by inhibition of APC proteosome function.
- Regulatory T cell suppression and anergy are differentially regulated by proinflammatory cytokines produced by TLR-activated dendritic cells.
- New developments in experimental models of inflammatory bowel disease.
- Gene disruption and immunity in experimental colitis.
- Immunology. T-bet or not T-bet.
- IFN-gamma-inducible chemokines enhance adaptive immunity and colitis.
- Restricted clonal expression of IL-2 by naive T cells reflects differential dynamic interactions with dendritic cells.
- Psoriasiform dermatitis susceptibility in Itgb2(tm1Bay) PL/J mice requires low-level CD18 expression and at least two additional loci for progression to severe disease.
- Delayed lupus onset in (NZB x NZW)F1 mice expressing a human C-reactive protein transgene.
- Preferential accumulation of antigen-specific effector CD4 T cells at an antigen injection site involves CD62E-dependent migration but not local proliferation.
- Bacterial-reactive T regulatory cells inhibit pathogenic immune responses to the enteric flora.
- Cutting-edge technology: IV. Genomic engineering for studies of the gastrointestinal tract in mice.
- Enhancement of MHC class II-restricted responses by receptor-mediated uptake of peptide antigens.
- Vitamin A enhances in vitro Th2 development via retinoid X receptor pathway.
- T helper 1 and T helper 2 cells are pathogenic in an antigen-specific model of colitis.
- Inhibition of T cell responses by transferrin-coupled competitor peptides.
- Diversity in the contribution of interleukin-10 to T-cell-mediated immune regulation.
- The central role of the Th17 lineage in regulating the inflammatory/autoimmune axis.
- Memory CD4 T cells emerge from effector T-cell progenitors.
- Immunology: narcissistic helpers.
- Transforming growth factor-beta induces development of the T(H)17 lineage.
- T-cell subsets: the more the merrier.
- Expanding the effector CD4 T-cell repertoire: the Th17 lineage.
- Experimental models of inflammatory bowel disease reveal innate, adaptive, and regulatory mechanisms of host dialogue with the microbiota.
- Efficient adenovirus-mediated gene transfer into primary T cells and thymocytes in a new coxsackie/adenovirus receptor transgenic model.
- Development of dermatitis in CD18-deficient PL/J mice is not dependent on bacterial flora, and requires both CD4+ and CD8+ T lymphocytes.
- Imaging CD8+ T cell dynamics in vivo using a transgenic luciferase reporter.
- Heterogeneity of single cell cytokine gene expression in clonal T cell populations.
- Single cell analysis of cytokine gene coexpression during CD4+ T-cell phenotype development.
- Visualization of antigen-specific T cell activation and cytokine expression in vivo.
- Single-cell analyses of CD4+ T cells from alpha beta T cell receptor-transgenic mice: a distinct mucosal cytokine phenotype in the absence of transgene-specific antigen.
- Heterogeneity in the clonal T cell response. Implications for models of T cell activation and cytokine phenotype development.
- Memory/effector T cells in TCR transgenic mice develop via recognition of enteric antigens by a second, endogenous TCR.
- Interleukin-2 expression by a subpopulation of primary T cells is linked to enhanced memory/effector function.
- Differential expression of Fas ligand in Th1 and Th2 cells is regulated by early growth response gene and NF-AT family members.
- Unexpected characteristics of the IFN-gamma reporters in nontransformed T cells.
- Differential activation of antigen-stimulated suicide and cytokine production pathways in CD4+ T cells is regulated by the antigen-presenting cell.
- Early upregulation of T cell IL-10 production plays an important role in oral tolerance induction.
- Nonopiate active proenkephalin-derived peptides are secreted by T helper cells.
- Immuno-bacterial homeostasis in the gut: new insights into an old enigma.
- Regional differences in L-selectin expression in murine intestinal lymphocytes.
- Colitis induced by enteric bacterial antigen-specific CD4+ T cells requires CD40-CD40 ligand interactions for a sustained increase in mucosal IL-12.
- Effector and suppressor roles for LFA-1 during the development of experimental autoimmune encephalomyelitis.
- The mucosal adjuvanticity of cholera toxin involves enhancement of costimulatory activity by selective up-regulation of B7.2 expression.
- Free radical-producing myeloid-derived regulatory cells: potent activators and suppressors of lung inflammation and airway hyperresponsiveness.
- Experimental models to study molecular mechanisms underlying intestinal inflammation.
- T helper cell subsets require the expression of distinct costimulatory signals by antigen-presenting cells.
- Membrane IL-1: a key protein in antigen presentation.
- T cell induction of macrophage IL-1 during antigen presentation. Characterization of a lymphokine mediator and comparison of TH1 and TH2 subsets.
- Peripheral tolerance to allogeneic class II histocompatibility antigens expressed in transgenic mice: evidence against a clonal-deletion mechanism.
- T cells of staphylococcal enterotoxin B-tolerized autoimmune MRL-lpr/lpr mice require co-stimulation through the B7-CD28/CTLA-4 pathway for activation and can be reane...
Schools
University Of Florida College Of Medicine
Barnes Jewish Hospital S Campus
Doctors Specialties
- Anatomic Pathology
- Pathology
Accepted Insurances
- Blue Card PPO
- Dental Blue 100/200/300
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Education
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Washington University - Barnes-Jewish Hospital -
University of Florida College of Medicine
Hospital
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University of Alabama Hospital -
Birmingham Veterans Affairs Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1598779761 |
| Last Name Of The Provider | WEAVER |
| First Name Of The Provider | CASEY |
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