Assistant Professor, Neurology, Washington University School of Medicine
Neurology
Dr. Conrad C. Weihl, MD
|
|
Give Review
Doctor Conrad C. Weihl, MD works in the field of Neurology. He went to University of Chicago Pritzker School of Medicine. University of Chicago Pritzker School of Medicine is ranked 10/19 in Research/PrimaryCare. He received 1 awards: "CMS Meaningful Use Stage 1 Certification". Dr. Conrad C. Weihl is also a published doctor. He has 49 paper published. The most recent paper is: Gene therapy for cerebrovascular disease. Conrad Weihl is registered with Medicare.
Publications
- Targeted sequencing and identification of genetic variants in sporadic inclusion body myositis.
- Autophagic vacuolar pathology in desminopathies.
- The VCP/p97 system at a glance: connecting cellular function to Disease pathogenesis.
- Myopathy-Causing Mutations in an Hsp40 Chaperone Disrupt Processing of Specific Client Conformers.
- Mutations in the Matrin 3 gene cause familial amyotrophic lateral sclerosis.
- Rapamycin nanoparticles target defective autophagy in Muscular dystrophy to enhance both strength and cardiac function.
- Increased autophagy accelerates colchicine-induced muscle Toxicity.
- Prion-like nuclear aggregation of TDP-43 during heat shock is regulated by HSP40/70 chaperones.
- p62 expression and autophagy in αB-crystallin R120G mutant knock-in mouse model of hereditary cataract.
- Motor neuron involvement in multisystem proteinopathy: Implications for ALS.
- Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS.
- Rapamycin-induced autophagy aggravates pathology and weakness in a mouse model of VCP-associated myopathy.
- Monitoring Autophagy in the Treatment of Protein Aggregate Diseases: Steps Toward Identifying Autophagic Biomarkers.
- mTOR dysfunction contributes to vacuolar pathology and weakness in valosin-containing protein associated inclusion body myopathy.
- Another VCP interactor: NF is enough.
- An aggregation sensing reporter identifies leflunomide and teriflunomide as polyglutamine aggregate inhibitors.
- Endolysosomal sorting of ubiquitylated caveolin-1 is regulated by VCP and UBXD1 and impaired by VCP Disease mutations.
- A knock-in mouse model for the R120G mutation of αB-crystallin recapitulates human hereditary myopathy and cataracts.
- Mitochondrial pathology in immune and Inflammatory myopathies.
- Quantitation of "autophagic flux" in mature skeletal muscle.
- Interaction with polyglutamine aggregates reveals a Q/N-rich domain in TDP-43.
- Quantitation of selective autophagic protein aggregate degradation in vitro and in vivo using luciferase reporters.
- p97/VCP at the intersection of the autophagy and the ubiquitin proteasome system.
- Valosin-containing protein (VCP) is required for autophagy and is disrupted in VCP Disease.
- Acute and bilateral blindness due to optic Neuropathy associated with copper deficiency.
- Impaired protein aggregate handling and clearance underlie the pathogenesis of p97/VCP-associated Disease.
- Motor neuron Disease associated with copper deficiency.
- Valproate may improve strength and function in patients with type III/IV spinal muscle atrophy.
- Novel GNE mutations in two phenotypically distinct HIBM2 patients.
- Valosin containing protein associated fronto-temporal lobar degeneration: clinical presentation, pathologic features and pathogenesis.
- Sporadic inclusion body myositis: possible pathogenesis inferred from biomarkers.
- Inclusion body myopathy-associated mutations in p97/VCP impair endoplasmic reticulum-associated degradation.
- Inclusion body myopathy, Paget''s Disease of the bone and fronto-temporal dementia: a disorder of autophagy.
- Myosin binding protein C1: a novel gene for autosomal dominant distal arthrogryposis type 1.
- Valosin-containing protein Disease: inclusion body myopathy with Paget''s Disease of the bone and fronto-temporal dementia.
- TDP-43 accumulation in inclusion body myopathy muscle suggests a common pathogenic mechanism with frontotemporal dementia.
- Inflammatory myopathies with mitochondrial pathology and protein aggregates.
- Transgenic expression of inclusion body myopathy associated mutant p97/VCP causes weakness and ubiquitinated protein inclusions in mice.
- Novel VCP mutations in inclusion body myopathy associated with Paget Disease of bone and frontotemporal dementia.
- Mutant presenilin-1 induces apoptosis and downregulates Akt/PKB.
- Processing of wild-type and mutant familial Alzheimer's Disease-associated presenilin-1 in cultured neurons.
- Alternative translation initiation of Theiler's murine encephalomyelitis virus.
- Creutzfeldt-Jakob Disease, new variant creutzfeldt-jakob Disease, and bovine spongiform encephalopathy.
- The role of beta-catenin stability in mutant PS1-associated apoptosis.
- Effect of adenovirus-mediated nitric oxide synthase gene transfer on vasospasm after experimental subarachnoid hemorrhage.
- Adenoviral gene transfer of nitric oxide synthase increases Cerebral blood flow in rats.
- [Pulse synchronous tinnitus as the only symptom of fibroMuscular dysplasia of cervical arteries].
- Gene therapy for cerebrovascular Disease.
Schools
U Of Chgo Div Of Bio Sci Pritzker Sch Of Med
Conditions Treated
- Anterior Horn Disease
- Ataxia
- Cerebral Artery Thrombosis
- Chronic Inflammatory Demyelinating Polyneuritis
- View All
Doctors Specialties
- Neurology
- Neuromuscular Medicine
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Anthem Blue Cross Blue Shield PPO
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Coventry Health Care of Missouri PPO
- First Health (Coventry Health Care) PPO
- Frontpath Health Coalition
- HealthLink PPO
- Homestate Health Plan
- Choice Care PPO
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- CMS Meaningful Use Stage 1 Certification
Education
-
Washington University -
University of Chicago Pritzker School of Medicine
Hospital
-
Barnes-Jewish Hospital
Drug Facts
| NPI NUMBER | 1437175759 | |
| NPPES Provider LastName | WEIHL | |
| NPPES Provider FirstName | CONRAD | |
| NPPES Provider ZIPCode | 631101032 | |
| NPPES Provider State | MO | |
| Specialty Description | Neurology | |
| Total Claim Count | 163.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1437175759 |
| Last Name Of The Provider | WEIHL |
| First Name Of The Provider | CONRAD |
| View All |
Similar Doctors
|
|
Andrew Findlay
 This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Craig M. Zaidman, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. James M. Goldring, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. John M. Zempel, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Steven R. Brenner, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Kristin Gehrking, MD |
|
|
Dr. Cindy V. Ly VI, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Poh-Hin Lim, MD |
|
|
Dr. Brenton A. Wright, MD
This Doctor is OpenDoctor Recommended.
|