Professor, Medicine, University of Minnesota Medical School
Pulmonology
Dr. David H. Ingbar, MD
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Doctor David H. Ingbar, MD is an expert in the field of Pulmonology. He attended Yale University School of Medicine. Yale University School of Medicine is one of the top medical schools in the US. Yale University School of Medicine is ranked 7/57 in Research/PrimaryCare. He received awards:"America's Top Doctors", "CMS Meaningful Use Stage 1 Certification", "Whos Who in Medicine and Healthcare" and "Super Doctor". Doctor David H. Ingbar, MD is a published doctor and has 81 publications published. The lastest was: 'Adrenergic stimulation of Na+ transport across alveolar epithelial cells involves activation of apical Cl- channels.' David H. Ingbar accepts all Medicare patients.
Publications
- Identification of a Cell-of-Origin for Fibroblasts Comprising the Fibrotic Reticulum in Idiopathic Pulmonary Fibrosis.
- Protein Expression Profile of Rat Type Two Alveolar Epithelial Cells During Hyperoxic Stress and Recovery.
- Adult alveolar epithelial cells express multiple subtypes of voltage-gated K+ channels that are located in apical membrane.
- Thyroid hormone stimulates Na-K-ATPase activity and its plasma membrane insertion in rat alveolar epithelial cells.
- Dexamethasone stimulates transcription of the Na+-K+-ATPase beta1 gene in adult rat Lung epithelial cells.
- Gene insertion and long-term expression in Lung mediated by the Sleeping Beauty transposon system.
- Dexamethasone responsive element in the rat Na, K-ATPase beta1 gene coding region.
- The critical care crisis in the United States: a report from the profession.
- 3,3',5-Triiodo-L-thyronine up-regulation of Na,K-ATPase activity and cell surface expression in alveolar epithelial cells is Src kinase- and phosphoinositide 3-kinase-...
- Pulmonary edema clearance: juicing up the sodium pump.
- Academic and career development of Pulmonary and critical care physician-scientists.
- Developmental acquisition of T3-sensitive Na-K-ATPase stimulation by rat alveolar epithelial cells.
- Thyroid hormone rapidly stimulates alveolar Na,K-ATPase by activation of phosphatidylinositol 3-kinase.
- T3 increases Na-K-ATPase activity via a MAPK/ERK1/2-dependent pathway in rat adult alveolar epithelial cells.
- Triiodo-L-thyronine rapidly stimulates alveolar fluid clearance in normal and hyperoxia-injured Lungs.
- Cell-specific signal transduction pathways regulating Na+-K+-ATPase. Focus on "short-term effects of thyroid hormones on the Na+-K+-ATPase activity of chick embryo hep...
- Nongenomic actions of L-thyroxine and 3,5,3'-triiodo-L-thyronine. Focus on "L-Thyroxine vs. 3,5,3'-triiodo-L-thyronine and cell proliferation: activation of mitogen-ac...
- Hypoxia: an unusual cause with specific treatment.
- Forging a critical alliance: Addressing the research needs of the United States critical illness and injury community.
- Mechanisms of alveolar epithelial chloride absorption.
- Src kinase integrates PI3K/Akt and MAPK/ERK1/2 pathways in T3-induced Na-K-ATPase activity in adult rat alveolar cells.
- Medical directors as partners in creating healthy work environments.
- On the function of half-site reactivity: intersubunit NAD+-dependent activation of acyl-glyceraldehyde 3-phosphate dehydrogenase reduction by NADH.
- Coagulopathy caused by intrauterine dextran.
- Induction of fibroblast apoptosis by anti-CD44 antibody: implications for the treatment of fibroproliferative Lung disease.
- Developmental regulation of Na, K-ATPase in rat Lung.
- Effects of hyperoxia on type II cell Na-K-ATPase function and expression.
- Developmental changes of fetal rat Lung Na-K-ATPase after maternal treatment with dexamethasone.
- Integrin mediation of alveolar epithelial cell migration on fibronectin and type I collagen.
- The critical early proinflammatory events associated with idiopathic Pneumonia syndrome in irradiated murine allogeneic recipients are due to donor T cell infusion and...
- The sodium pump and oxidant stress: if only it were so simple.
- Cell density and antioxidant vitamins determine the effects of hyperoxia on proliferation and death of MDCK epithelial cells.
- Hyperoxic effects on alveolar sodium resorption and Lung Na-K-ATPase.
- Perfusion technique determines alveolar fluid resorption rate in the isolated perfused rat Lung.
- Regulation of Na-K-ATPase gene expression by hyperoxia in MDCK cells.
- The effects of hyperoxic injury and antioxidant vitamins on death and proliferation of human small airway epithelial cells.
- Oxidant effects on epithelial Na,K-ATPase gene expression and promoter function.
- Adrenergic stimulation of Na+ transport across alveolar epithelial cells involves activation of apical Cl- channels.
- High levels of peroxynitrite are generated in the Lungs of irradiated mice given cyclophosphamide and allogeneic T cells. A potential mechanism of injury after marrow ...
- Na(+)-K(+)-ATPase gene regulation by glucocorticoids in a fetal Lung epithelial cell line.
- Activated alveolar macrophage-derived nitric oxide predicts the development of Lung damage after marrow transplantation in mice.
- Hyperoxia upregulated Na,K-adenosine triphosphatase beta1 gene transcription.
- Hepatocyte growth factor stimulates migration of type II alveolar epithelial cells on the provisional matrix proteins fibronectin and fibrinogen.
- Hyperoxic Lung injury increases HOCl-modified Lung proteins and NA,K-adenosine triphosphatase nitrotyrosine content.
- Interactions of keratinocyte growth factor with a nitrating species after marrow transplantation in mice.
- Cl-channel activation is necessary for stimulation of Na transport in adult alveolar epithelial cells.
- KGF pretreatment decreases B7 and granzyme B expression and hastens repair in Lungs of mice after allogeneic BMT.
- Up-regulation of Na,K-ATPase beta 1 transcription by hyperoxia is mediated by SP1/SP3 binding.
- Mechanisms of repair and remodeling following acute Lung injury.
- Selectivity properties of a Na-dependent amino acid cotransport System in adult alveolar epithelial cells.
- Cyclophosphamide prevents Systemic keratinocyte growth factor-induced up-regulation of surfactant protein A after allogeneic transplant in mice.
- Human surfactant protein a suppresses T cell-dependent inflammation and attenuates the manifestations of idiopathic Pneumonia syndrome in mice.
- Adrenergic regulation of ion transport across adult alveolar epithelial cells: effects on Cl- channel activation and transport function in cultures with an apical air ...
- gamma-Glutamyl transpeptidase is a polarized alveolar epithelial membrane protein.
- Induction of endothelial cell apoptosis by TNF alpha: modulation by inhibitors of protein synthesis.
- Alveolar epithelial cells regulate the induction of endothelial cell apoptosis.
- Diagnosis and management of acute Lung injury.
- Repair after acute Lung injury.
- Hyperoxia increases active alveolar Na+ resorption in vivo and type II cell Na,K-ATPase in vitro.
- Massive hemoptysis. Assessment and management.
- Role of mesenchymal cell death in Lung remodeling after injury.
- Cell fusion to study nuclear-cytoplasmic interactions in endothelial cell apoptosis.
- Integrin mediation of type II cell adherence to provisional matrix proteins.
- Hyperoxia enhances expression of gamma-glutamyl transpeptidase and increases protein S-glutathiolation in rat Lung.
- Terbutaline stimulates alveolar fluid resorption in hyperoxic Lung injury.
- The effect of food deprivation of the peripheral metabolism of thyroxine in rats.
- A universal precautions monitoring System adaptable to any health care department.
- Upregulation of rat Lung Na-K-ATPase during hyperoxic injury.
- Response of rat type II pneumocyte Na,K-ATPase to hyperoxic injury.
- Polarized distribution of Na+,K+-ATPase in giant cells elicited in vivo and in vitro.
- An in vitro model for polymorphonuclear-leukocyte-induced injury to an extracellular matrix. Relative contribution of oxidants and elastase to fibronectin release from...
- Acute Respiratory failure.
- Pulmonary sequelae and lung repair in survivors of the adult respiratory distress syndrome.
- Repopulation of a human alveolar matrix by adult rat type II pneumocytes in vitro. A novel System for type II pneumocyte culture.
- Fatal infective endocarditis in a young adult with persistent truncus arteriosus and a bicuspid truncal valve.
- Pathophysiology and treatment of Sleep apnea.
- Bleomycin hydrolase activity in Pulmonary cells.
- Invited review: Lung edema clearance: role of Na(+)-K(+)-ATPase.
- Basolateral Cl- transport is stimulated by terbutaline in adult rat alveolar epithelial cells.
- Proteomic profiles in acute Respiratory distress syndrome differentiates survivors from non-survivors.
Schools
Harvard Medical School
University Wa
Yale University
Procedures Preformed
- Bronchoscopy
- Continuous Positive Airway Pressure (CPAP)
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Bronchiectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Awards
- America's Top Doctors
- CMS Meaningful Use Stage 1 Certification
- Whos Who in Medicine and Healthcare
- Super Doctor
Education
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Yale University School of Medicine -
University of Washington -
Harvard Medical School
Hospital
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University of Minnesota Medical Center, Fairview
Drug Facts
| NPI NUMBER | 1063446557 | |
| NPPES Provider LastName | INGBAR | |
| NPPES Provider FirstName | DAVID | |
| NPPES Provider ZIPCode | 55455 | |
| NPPES Provider State | MN | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 375.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1063446557 |
| Last Name Of The Provider | INGBAR |
| First Name Of The Provider | DAVID |
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