Physician
Nephrology
Dr. Fuad N. Ziyadeh, MD
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Fuad Ziyadeh practices Nephrology. He received 3 awards: "Fellow (ASNF)", "Member" and "Fellow". Fuad N. Ziyadeh, MD is a published doctor and has 101 publications published. The lastest was: 'Estradiol reverses TGF-beta1-stimulated type IV collagen gene transcription in murine mesangial cells.'
Publications
- Vascular endothelial growth factor and diabetic nephropathy.
- Steroid treatment of drug-induced acute interstitial nephritis.
- The utility of the transtubular potassium gradient in the evaluation of hyperkalemia.
- Interference with TGF-beta signaling by Smad3-knockout in mice limits diabetic glomerulosclerosis without affecting albuminuria.
- Taurine intestinal absorption and renal excretion test in diabetic patients: a pilot study.
- Blockade of vascular endothelial growth factor signaling ameliorates diabetic albuminuria in mice.
- Amadori-modified glycated serum proteins and accelerated atherosclerosis in diabetes: pathogenic and therapeutic implications.
- Assessment of 115 candidate genes for diabetic nephropathy by transmission/disequilibrium test.
- Evidence linking glycated albumin to altered glomerular nephrin and VEGF expression, proteinuria, and diabetic nephropathy.
- From the periphery of the glomerular capillary wall toward the center of disease: podocyte injury comes of age in diabetic nephropathy.
- Albumin up-regulates the type II transforming growth factor-beta receptor in cultured proximal tubular cells.
- Podocyte-derived vascular endothelial growth factor mediates the stimulation of alpha3(IV) collagen production by transforming growth factor-beta1 in mouse podocytes.
- Cultured tubule cells from TGF-beta1 null mice exhibit impaired hypertrophy and fibronectin expression in high glucose.
- Mediators of diabetic renal disease: the case for tgf-Beta as the major mediator.
- Diabetic nephropathy and transforming growth factor-beta: transforming our view of glomerulosclerosis and fibrosis build-up.
- Why should an angiogenic factor modulate tubular structure in diabetic nephropathy? Some answers, more questions.
- Overview: combating diabetic nephropathy.
- Reversibility of established diabetic glomerulopathy by anti-TGF-beta antibodies in db/db mice.
- Smad pathway is activated in the diabetic mouse kidney and Smad3 mediates TGF-beta-induced fibronectin in mesangial cells.
- Effects of high glucose and TGF-beta1 on the expression of collagen IV and vascular endothelial growth factor in mouse podocytes.
- Inhibiting albumin glycation in vivo ameliorates glomerular overexpression of TGF-beta1.
- Role of basic fibroblast growth factor-2 in epithelial-mesenchymal transformation.
- Leptin and renal disease.
- Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing S...
- Inhibiting albumin glycation ameliorates diabetic nephropathy in the db/db mouse.
- Stimulation of TGF-beta type II receptor by high glucose in mouse mesangial cells and in diabetic kidney.
- The urine/plasma electrolyte ratio: a predictive guide to water restriction.
- Therapy with antisense TGF-beta1 oligodeoxynucleotides reduces kidney weight and matrix mRNAs in diabetic mice.
- Hepatocyte growth factor: a regulator of extracellular matrix genes in mouse mesangial cells.
- Angiotensin converting-enzyme inhibitor treatment reduces glomerular p16INK4 and p27Kip1 expression in diabetic BBdp rats.
- GLUT1 and TGF-beta: the link between hyperglycaemia and diabetic nephropathy.
- Captopril-induced reduction of serum levels of transforming growth factor-beta1 correlates with long-term renoprotection in insulin-dependent diabetic patients.
- Angiotensin II inhibits inducible nitric oxide synthase in tubular MCT cells by a posttranscriptional mechanism.
- Angiotensin II stimulates expression of transforming growth factor beta receptor type II in cultured mouse proximal tubular cells.
- High glucose stimulates proliferation and collagen type I synthesis in renal cortical fibroblasts: mediation by autocrine activation of TGF-beta.
- Leptin stimulates proliferation and TGF-beta expression in renal glomerular endothelial cells: potential role in glomerulosclerosis [seecomments].
- Molecular mechanisms of diabetic renal hypertrophy.
- Angiotensin II induces alpha3(IV) collagen expression in cultured murine proximal tubular cells.
- Favorable treatment outcome with neutralizing anti-transforming growth factor beta antibodies in experimental diabetic kidney disease.
- What is the role of decorin in diabetic kidney disease?
- Glycated albumin stimulation of PKC-beta activity is linked to increased collagen IV in mesangial cells.
- Estradiol reverses TGF-beta1-stimulated type IV collagen gene transcription in murine mesangial cells.
- Increased decorin mRNA in diabetic mouse kidney and in mesangial and tubular cells cultured in high glucose.
- Transcriptional activation of transforming growth factor-beta1 in mesangial cell culture by high glucose concentration.
- [Current questions concerning the pathomechanism of diabetic nephropathy].
- The renal TGF-beta system in the db/db mouse model of diabetic nephropathy.
- Glomerular expression of p27Kip1 in diabetic db/db mouse: role of hyperglycemia.
- Potential role of TGF-beta in diabetic nephropathy.
- Glycated albumin stimulates fibronectin gene expression in glomerular mesangial cells: involvement of the transforming growth factor-beta system.
- Serum-stimulated alpha 1 type IV collagen gene transcription is mediated by TGF-beta and inhibited by estradiol.
- Activation of glomerular mesangial cells by hepatocyte growth factor through tyrosine kinase and protein kinase C.
- Language guiding therapy: the case of dehydration versus volume depletion.
- High glucose stimulates expression of p27Kip1 in cultured mouse mesangial cells: relationship to hypertrophy.
- Renal tubular hypertrophy induced by angiotensin II.
- Involvement of transforming growth factor-beta and its receptors in the pathogenesis of diabetic nephrology.
- Angiotensin II stimulates expression of the chemokine RANTES in rat glomerular endothelial cells. Role of the angiotensin type 2 receptor.
- Biochemical events and cytokine interactions linking glucose metabolism to the development of diabetic nephropathy.
- RAGE mRNA expression in the diabetic mouse kidney.
- Increased renal production of transforming growth factor-beta1 in patients with type II diabetes.
- Expression of apoptosis-regulatory genes in renal proximal tubular epithelial cells exposed to high ambient glucose and in diabetic kidneys.
- [Current issues in the diagnosis and therapy of diabetic nephropathy].
- Early and advanced non-enzymatic glycation products in the pathogenesis of diabetic kidney disease.
- The role of angiotensin II in diabetic nephropathy: emphasis on nonhemodynamic mechanisms.
- Cyclosporin A induces transcription of transforming growth factor beta in a cultured murine proximal tubular cell line.
- Role of Amadori-modified nonenzymatically glycated serum proteins in the pathogenesis of diabetic nephropathy.
- Evolution of renal function abnormalities in the db/db mouse that parallels the development of human diabetic nephropathy.
- The use of neutralizing antibodies to demonstrate the role of transforming growth factor-beta and Amadori-glycated albumin as mediators of experimental diabetic kidney...
- Update on pathogenesis, markers and management of diabetic nephropathy.
- Significance of tubulointerstitial changes in diabetic renal disease.
- Diabetes and hypertension. Australian Diabetes Society position statement.
- Angiotensin II is mitogenic for cultured rat glomerular endothelial cells.
- Neutralization of TGF-beta by anti-TGF-beta antibody attenuates kidney hypertrophy and the enhanced extracellular matrix gene expression in STZ-induced diabetic mice.
- Angiotensin II-stimulated expression of transforming growth factor beta in renal proximal tubular cells: attenuation after stable transfection with the c-mas oncogene.
- Albumin modified by Amadori glucose adducts activates mesangial cell type IV collagen gene transcription.
- Role of protein kinase C and cyclic AMP/protein kinase A in high glucose-stimulated transcriptional activation of collagen alpha 1 (IV) in glomerular mesangial cells.
- Facilitative glucose transport proteins and sodium-glucose co-transporters in the kidney.
- Renal tubular basement membrane and collagen type IV in diabetes mellitus.
- The transforming growth factor-beta system and the kidney.
- Effects of glycated albumin on mesangial cells: evidence for a role in diabetic nephropathy.
- The extracellular matrix in diabetic nephropathy.
- Polyol pathway mediates high glucose-induced collagen synthesis in proximal tubule.
- Amadori glucose adducts modulate mesangial cell growth and collagen gene expression.
- Stimulation of collagen gene expression and protein synthesis in murine mesangial cells by high glucose is mediated by autocrine activation of transforming growth fact...
- Role of transforming growth factor beta in diabetic nephropathy.
- The emerging role of transforming growth factor-beta in kidney diseases.
- Renal hypertrophy is associated with upregulation of TGF-beta 1 gene expression in diabetic BB rat and NOD mouse.
- Glycated albumin modified by Amadori adducts modulates aortic endothelial cell biology.
- ANG II is a mitogen for a murine cell line isolated from medullary thick ascending limb of Henle's loop.
- Growth arrest of a murine mesangial cell line by transforming growth factor beta 1 is associated with inhibition of mitogen-induced Ca2+ mobilization.
- Prevention of diabetic nephropathy in db/db mice with glycated albumin antagonists. A novel treatment strategy.
- Angiotensin II-induced hypertrophy of cultured murine proximal tubular cells is mediated by endogenous transforming growth factor-beta.
- The diabetic renal tubulointerstitium.
- Atrial natriuretic peptide stimulates the expression of transforming growth factor-beta in cultured murine mesangial cells: relationship to suppression of proliferation.
- Mediators of hyperglycemia and the pathogenesis of matrix accumulation in diabetic renal disease.
- Hyperglycemia and diabetic kidney disease. The case for transforming growth factor-beta as a key mediator.
- Enhanced expression of inducible nitric oxide synthase in murine macrophages and glomerular mesangial cells by elevated glucose levels: possible mediation via protein ...
- PKC and high glucose stimulate collagen alpha 1 (IV) transcriptional activity in a reporter mesangial cell line.
- Role of transforming growth factor-beta in diabetic glomerulosclerosis and renal hypertrophy.
- Fractional excretion of chloride in prerenal azotemia.
- Thiazides stimulate calcium absorption in urinary bladder of winter flounder.
Schools
American University Of Beirut Faculty Of Medicine
American University Beirut Med Center
Hospital University Pa
Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- Fellow (ASNF)
- Member
- Fellow
Education
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University of Pennsylvania Health System -
American University of Beirut Faculty of Medicine
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