Physician
Pulmonology
Dr. Gregory Cieslewicz, MD
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Gregory Cieslewicz is an expert in the field of Pulmonology. He went to Wayne State University School of Medicine. Wayne State University School of Medicine is ranked 63 in research and 85 in primary care medicine Doctor Gregory Cieslewicz, MD is also a published doctor. He has 17 publication published. The most recent publication is: Local treatment with IL-12 is an effective inhibitor of airway hyperresponsiveness and lung eosinophilia after airway challenge in sensitized mice.
Publications
- [Effect of loratadine--selective antagonist of histamine (H1) receptor--on allergen-induced bronchoconstriction in atopic Asthmatics].
- Transfer of the enhancing effect of Respiratory syncytial virus infection on subsequent allergic airway sensitization by T lymphocytes.
- CD8 T cells are essential in the development of respiratory syncytial virus-induced Lung eosinophilia and airway hyperresponsiveness.
- IL-5 and eosinophils are essential for the development of airway hyperresponsiveness following acute Respiratory syncytial virus infection.
- Eotaxin-1-deficient mice develop airway eosinophilia and airway hyperresponsiveness.
- Anti-interleukin 5 but not anti-IgE prevents airway inflammation and airway hyperresponsiveness.
- Interleukin (IL)-5 but not immunoglobulin E reconstitutes airway inflammation and airway hyperresponsiveness in IL-4-deficient mice.
- Local treatment with IL-12 is an effective inhibitor of airway hyperresponsiveness and Lung eosinophilia after airway challenge in sensitized mice.
- The Failure of STAT6-deficient mice to develop airway eosinophilia and airway hyperresponsiveness is overcome by interleukin-5.
- Temporal association between airway hyperresponsiveness and airway eosinophilia in ovalbumin-sensitized mice.
- A murine IL-4 receptor antagonist that inhibits IL-4- and IL-13-induced responses prevents antigen-induced airway eosinophilia and airway hyperresponsiveness.
- The late, but not early, Asthmatic response is dependent on IL-5 and correlates with eosinophil infiltration.
- Anti-CD86 (B7.2) treatment abolishes allergic airway hyperresponsiveness in mice.
- Extensive eosinophil degranulation and peroxidase-mediated oxidation of airway proteins do not occur in a mouse ovalbumin-challenge model of Pulmonary inflammation.
- Eosinophil major basic protein-1 does not contribute to allergen-induced airway pathologies in mouse models of Asthma.
- Early phase bronchoconstriction in the mouse requires allergen-specific IgG.
Schools
Lodzi
Wayne State University
University of Washington
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asbestosis
- Asthma
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Doctors Specialties
- Pulmonology
Accepted Insurances
Education
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National Institutes of Health Clinical Center -
University of Washington -
Wayne State University School of Medicine -
Medical University of Lodz
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