Associate Professor of Neurology
Neurology
Dr. Harold Moses Jr., MD
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Harold Moses Jr. works in the field of Neurology. He researched medicine at University of North Carolina at Chapel Hill School of Medicine. University of North Carolina at Chapel Hill School of Medicine is one of the top medical schools in the US. University of North Carolina at Chapel Hill School of Medicine is ranked 22 in research and 2 in primary care medicine He received awards:"Elected Member". Harold Moses has written publications. He has 66 publications published with PubMed. The lastest publication is: 'STAT3 Mediated Remodeling of the Tumor Microenvironment Results in Enhanced Tumor Drug Delivery in a Mouse Model of Pancreatic Cancer.' He accepts Medicare payments and is listed with Medicare.gov.
Publications
- Small molecule inhibitor of the bone morphogenetic protein pathway DMH1 reduces ovarian Cancer cell growth.
- STAT3 Mediated Remodeling of the Tumor Microenvironment Results in Enhanced Tumor Drug Delivery in a Mouse Model of Pancreatic Cancer.
- Age- and pregnancy-associated DNA methylation changes in mammary epithelial cells.
- Role of TGF-β Signaling in Generation of CD39+CD73+ Myeloid Cells in Tumors.
- Concerted loss of TGFβ-mediated proliferation control and E-cadherin disrupts epithelial homeostasis and causes oral squamous cell carcinoma.
- Inhibition of BMP signaling suppresses metastasis in mammary Cancer.
- The roles of TGFβ in the tumour microenvironment.
- Diagnostic and therapeutic aspects of Hashimoto's encephalopathy.
- Stromally Derived Lysyl Oxidase Promotes Metastasis of Transforming Growth Factor-β-Deficient Mouse Mammary Carcinomas.
- Turn Off the IDO: Will Clinical Trials Be Successful?
- Lack of transforming growth factor-β signaling promotes collective Cancer cell invasion through tumor-stromal crosstalk.
- Deletion of TGF-β signaling in myeloid cells enhances their anti-tumorigenic properties.
- Pathway Analyses Identify TGFBR2 as Potential Breast Cancer Susceptibility Gene: Results from a Consortium Study among Asians.
- p120-catenin is Essential for terminal end bud function and mammary morphogenesis.
- ?-Catenin and K-RAS Synergize to Form Primitive Renal Epithelial Tumors with Features of Epithelial Wilms' Tumors.
- Inhibiting Cxcr2 disrupts tumor-stromal interactions and improves survival in a mouse model of pancreatic ductal adenocarcinoma.
- Myeloid Suppressor Cells Regulate the Lung Environment--Response.
- Altered prostate epithelial development in mice lacking the androgen receptor in stromal fibroblasts.
- Breast Cancer Special Feature: Disruption of bone morphogenetic protein receptor 2 (BMPR2) in mammary tumors promotes metastases through cell autonomous and paracrine ...
- Analysis of transforming growth factor β receptor expression and signaling in higher grade Meningiomas.
- TRAIL and interferon-alpha act synergistically to induce renal cell carcinoma apoptosis.
- TGF-beta receptor deletion in the renal collecting System exacerbates fibrosis.
- Gr-1+CD11b+ myeloid cells tip the balance of immune protection to tumor promotion in the premetastatic lung.
- Meharry Medical College-Vanderbilt-Ingram Cancer Center partnership: its history and role in Cancer health disparities research.
- Gain or loss of TGFbeta signaling in mammary carcinoma cells can promote metastasis.
- Abrogation of TGF-beta signaling enhances chemokine production and correlates with prognosis in human breast Cancer.
- GFAP-Cre-mediated activation of oncogenic K-ras results in expansion of the subventricular zone and infiltrating glioma.
- TGF-beta promotes cell death and suppresses lactation during the second stage of mammary involution.
- Transforming growth factor beta: tumor suppressor or promoter? Are host immune cells the answer?
- Identification of novel Smad2 and Smad3 associated proteins in response to TGF-beta1.
- Transforming growth factor-beta signaling-deficient fibroblasts enhance hepatocyte growth factor signaling in mammary carcinoma cells to promote scattering and invasion.
- Transforming growth factor-beta (TGF-beta) and TGF-beta-associated kinase 1 are required for R-Ras-mediated transformation of mammary epithelial cells.
- Transforming growth factor-beta regulates mammary carcinoma cell survival and interaction with the adjacent microenvironment.
- Abrogation of TGF beta signaling in mammary carcinomas recruits Gr-1+CD11b+ myeloid cells that promote metastasis.
- Signaling pathways regulating TC21-induced tumorigenesis.
- A delicate balance: TGF-beta and the tumor microenvironment.
- TGF-beta signaling is Essential for joint morphogenesis.
- Enhanced hepatocyte growth factor signaling by type II transforming growth factor-beta receptor knockout fibroblasts promotes mammary tumorigenesis.
- A tale of two proteins: differential roles and regulation of Smad2 and Smad3 in TGF-beta signaling.
- Inhibition of TGF-beta with neutralizing antibodies prevents radiation-induced acceleration of metastatic Cancer progression.
- Epidermal growth factor receptor plays a significant role in hepatocyte growth factor mediated biological responses in mammary epithelial cells.
- Transcriptional cooperation between the transforming growth factor-beta and Wnt pathways in mammary and intestinal tumorigenesis.
- Aggressive pancreatic ductal adenocarcinoma in mice caused by pancreas-specific blockade of transforming growth factor-beta signaling in cooperation with active Kras e...
- Transforming growth factor beta receptor type II inactivation induces the malignant transformation of intestinal neoplasms initiated by Apc mutation.
- Oncogenic function of a novel WD-domain protein, STRAP, in human carcinogenesis.
- Effect of IGF-I in the chondrogenesis of bone marrow mesenchymal stem cells in the presence or absence of TGF-beta signaling.
- Under pressure: stromal fibroblasts change their ways.
- Genomic and proteomic analysis of mammary tumors arising in transgenic mice.
- Proliferation of estrogen receptor-alpha-positive mammary epithelial cells is restrained by transforming growth factor-beta1 in adult mice.
- Keratin 19 gene drives Cre recombinase expression throughout the early postimplantation mouse embryo.
- Activin receptor-like kinase 2 and Smad6 regulate epithelial-mesenchymal transformation during cardiac valve formation.
- Effect of conditional knockout of the type II TGF-beta receptor gene in mammary epithelia on mammary gland development and polyomavirus middle T antigen induced tumor ...
- Conditional deletion of the TGF-beta type II receptor in Col2a expressing cells results in defects in the axial skeleton without alterations in chondrocyte differentia...
- Activation of the Erk pathway is required for TGF-beta1-induced EMT in vitro.
- Transforming growth factor beta receptor type II inactivation promotes the establishment and progression of colon Cancer.
- TGF-beta signaling in fibroblasts modulates the oncogenic potential of adjacent epithelia.
- TGF-beta-induced RhoA and p160ROCK activation is involved in the inhibition of Cdc25A with resultant cell-cycle arrest.
- Increased malignancy of Neu-induced mammary tumors overexpressing active transforming growth factor beta1.
- Transgenic mice expressing a dominant-negative mutant type II transforming growth factor-beta receptor exhibit impaired mammary development and enhanced mammary tumor ...
- The loss of TGF-beta signaling promotes prostate Cancer metastasis.
- Rapamycin potentiates transforming growth factor beta-induced growth arrest in nontransformed, oncogene-transformed, and human Cancer cells.
- Transforming growth factor beta mimetics: Discovery of 7-[4-(4-cyanophenyl)phenoxy]-heptanohydroxamic acid, a biaryl hydroxamate inhibitor of histone deacetylase.
- Conditional inactivation of the TGF-beta type II receptor using Cre:Lox.
- TGFβ signaling in myeloid cells regulates mammary carcinoma cell invasion through fibroblast interactions.
- Bone morphogenetic proteins stimulate mammary fibroblasts to promote mammary carcinoma cell invasion.
Schools
University Of North Carolina Chapel Hill School Of Medicine
Mayo Grad School MedMayo Fndn
University Of Nc Hosps
Procedures Preformed
- EEG (Electroencephalogram)
- Non-Cosmetic Chemodenervation
Conditions Treated
- Alzheimer's Disease
- Ataxia
- Brain and Nervous System Cancer (incl. Gliomas, Astrocytoma, Schwannoma, Medulloblastoma, Chordoma)
- Brain Aneurysm
- View All
Doctors Specialties
- Neurology
Accepted Insurances
Awards
- Elected Member
Education
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Mayo School of Graduate Medical Education -
University of North Carolina Hospitals -
University of North Carolina at Chapel Hill School of Medicine
Hospital
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Vanderbilt Hospital and Clinics
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