Professor, Dermatology, Emory University School of Medicine
Dermatology
Dr. Jack L. Arbiser, MD
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Doctor Jack L. Arbiser, MD works in the field of Dermatology. He attended Harvard Medical School. Harvard Medical School is one of the top medical schools in the US. Harvard Medical School is ranked 1/12 in Research/PrimaryCare. Jack Arbiser is a published physician. He published 101 publications, including: 'Seborrheic Keratoses: The Rodney Dangerfield of Skin lesions, and Why They Should Get Our Respect.' Doctor Jack L. Arbiser, MD accepts Medicare.
Publications
- Seborrheic Keratoses: The Rodney Dangerfield of Skin lesions, and Why They Should Get Our Respect.
- Designing a broad-spectrum integrative approach for Cancer prevention and treatment.
- Tris DBA palladium overcomes hypoxia-mediated drug resistance in multiple myeloma.
- Imipramine blue halts head and neck Cancer invasion through promoting F-box and leucine-rich repeat protein 14-mediated Twist1 degradation.
- Evasion of anti-growth signaling: a key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.
- Inactivation of NADPH Oxidases NOX4 and NOX5 Protects Human Primary Fibroblasts from Ionizing Radiation-Induced DNA Damage.
- NADPH oxidase 4 is a critical mediator in Ataxia telangiectasia disease.
- Hyperglycemia and redox status regulate RUNX2 DNA-binding and an angiogenic phenotype in endothelial Cells.
- The antioxidant paradox: what are antioxidants and how should they be used in a therapeutic context for Cancer.
- PHIPing Out: A Genetic Basis for Tumor Ulceration.
- Current approaches to the treatment of metastatic brain tumours.
- Honokiol inhibits epithelial-mesenchymal transition in breast Cancer cells by targeting signal transducer and activator of transcription 3/Zeb1/E-cadherin axis.
- Gentian Violet: a 19th century drug re-emerges in the 21st century.
- Honokiol inhibits androgen receptor activity in prostate Cancer cells.
- Identifying new small molecule anti-invasive compounds for glioma treatment.
- Selectivity, Binding Affinity, and Ionization State of Matrix Metalloproteinase Inhibitors.
- Combination therapy of imiquimod and gentian violet for cutaneous Melanoma metastases.
- Metabotropic glutamate receptor-dependent long-term depression is impaired due to elevated ERK signaling in the ??RG mouse model of tuberous sclerosis complex.
- Honokiol stimulates osteoblastogenesis by suppressing NF-?B activation.
- Beating the rap in angiogenesis.
- Fumarate esters as angiogenesis inhibitors: key to action in psoriasis?
- Fisetin: A Natural Fist against Melanoma?
- A Key Role for NOX4 in Epithelial Cell Death During Development of Lung Fibrosis.
- Novel antiangiogenic agents in dermatology.
- Disruption of the mitochondrial thioredoxin system as a Cell death mechanism of cationic triphenylmethanes.
- Peroxisome proliferation-associated control of reactive oxygen species sets melanocortin tone and feeding in diet-induced obesity.
- High level expression of angiopoietin-2 in human abscesses.
- Corrigendum: Peroxisome proliferation-associated control of reactive oxygen species sets melanocortin tone and feeding in diet-induced obesity.
- Effectiveness of gentian violet and similar products commonly used to treat pyodermas.
- A nonsteroidal alternative to impetiginized eczema in the emergency room.
- EC-specific chemotaxis receptor: a double-edged sword.
- Honokiol inhibits epidermal growth factor receptor signaling and enhances the antitumor effects of epidermal growth factor receptor inhibitors.
- Ulcerative granulomatous mycosis fungoides.
- Rapid improvement of nephrogenic systemic fibrosis with rapamycin therapy: possible role of phospho-70-ribosomal-S6 kinase.
- Translating cyclooxygenase signaling in patch heterozygote mice into a randomized clinical trial in basal Cell carcinoma.
- Disseminated pyoderma gangrenosum: role for vascular endothelial growth factor and hypoxia inducible factor-2.
- Tuberous sclerosis complex suppression in cerebellar development and medulloblastoma: separate regulation of mammalian target of rapamycin activity and p27 Kip1 locali...
- Fulvene-5 potently inhibits NADPH oxidase 4 and blocks the growth of endothelial tumors in mice.
- Gentian violet is safe.
- Secreted frizzle-related protein 2 stimulates angiogenesis via a calcineurin/NFAT signaling pathway.
- Melanomas reveal their nakedness: uncovered by interferon alfa.
- Honokiol, a multifunctional antiangiogenic and antitumor agent.
- Maspin expression, angiogenesis, prognostic parameters, and outcome in malignant Melanoma.
- Unexpected autocrine role of vascular endothelial growth factor in Squamous cell carcinoma.
- Efficacy of topical application of eosin for ulcerated hemangiomas.
- The hunting of the Snrk.
- Prox-1 promotes invasion of kaposiform hemangioendotheliomas.
- Immunosuppression may be present within condyloma acuminata.
- Solenopsin A, a venom alkaloid from the fire ant Solenopsis invicta, inhibits quorum-sensing signaling in Pseudomonas aeruginosa.
- Tris (dibenzylideneacetone) dipalladium, a N-myristoyltransferase-1 inhibitor, is effective against Melanoma growth in vitro and in vivo.
- A modified cysteinyl-labeling assay reveals reversible oxidation of protein tyrosine phosphatases in angiomyolipoma Cells.
- The natural product honokiol preferentially inhibits Cellular FLICE-inhibitory protein and augments death receptor-induced apoptosis.
- Honokiol suppresses survival signals mediated by Ras-dependent phospholipase D activity in human Cancer cells.
- Targeted therapy of oral hairy leukoplakia with gentian violet.
- Shb gene knockdown increases the susceptibility of SVR endothelial tumor Cells to apoptotic stimuli in vitro and in vivo.
- Application of angiogenesis to clinical dermatology.
- Efficacy of rapamycin in scleroderma: a case study.
- Angiogenesis in cutaneous Lesions of leprosy: implications for treatment.
- Why targeted therapy hasn't worked in advanced Cancer.
- Honokiol, a natural plant product, inhibits inflammatory signals and alleviates inflammatory arthritis.
- Honokiol, a natural plant product, inhibits the bone metastatic growth of human prostate Cancer cells.
- AKT1 overexpression in endothelial Cells leads to the development of cutaneous vascular malformations in vivo.
- Expression of the neural stem cell markers NG2 and L1 in human angiomyolipoma: are angiomyoLipomas neoplasms of stem cells?
- Overexpression of Akt converts radial growth Melanoma to vertical growth Melanoma.
- Solenopsin, the alkaloidal component of the fire ant (Solenopsis invicta), is a naturally occurring inhibitor of phosphatidylinositol-3-kinase signaling and angiogenesis.
- Curcumin as an inhibitor of angiogenesis.
- Mammalian target of rapamycin (mTOR) is activated in cutaneous vascular malformations in vivo.
- Presence of p16 hypermethylation and Epstein-Barr virus infection in transplant-associated hematolymphoid neoplasm of the Skin.
- The duality of angiogenesis: implications for therapy of human disease.
- Pharmacologic blockade of angiopoietin-2 is efficacious against model hemangiomas in mice.
- Honokiol potentiates apoptosis, suppresses osteoclastogenesis, and inhibits invasion through modulation of nuclear factor-kappaB activation pathway.
- Wilms tumor 1 expression present in most Melanomas but nearly absent in nevi.
- Carbazole is a naturally occurring inhibitor of angiogenesis and inflammation isolated from antipsoriatic coal tar.
- Facile purification of honokiol and its antiviral and cytotoxic properties.
- Combination therapy of doxycycline and topical tacrolimus for venous ulcers.
- Expression of Wilms tumor 1 gene distinguishes vascular malformations from proliferative endothelial Lesions.
- Apolipoprotein D is down-regulated during malignant transformation of neurofibromas.
- Naturally occurring proteasome inhibitors from mate tea (Ilex paraguayensis) serve as models for topical proteasome inhibitors.
- Implications of Epstein-Barr Virus (EBV)-induced carcinogenesis on cutaneous inflammation and carcinogenesis: evidence of recurring patterns of angiogenesis and signal...
- Malignant transformation of human Cells by constitutive expression of platelet-derived growth factor-BB.
- Transgenic expression of dominant negative tuberin through a strong constitutive promoter results in a tissue-specific tuberous sclerosis phenotype in the Skin and brain.
- PSK and Trx80 inhibit B-Cell growth in EBV-infected cord blood mononuclear Cells through T Cells activated by the monocyte products IL-15 and IL-12.
- gli-1 Oncogene is highly expressed in granulomatous Skin disorders, including sarcoidosis, granuloma annulare, and necrobiosis lipoidica diabeticorum.
- Rapamycin (sirolimus) as a steroid-sparing agent in dermatomyositis.
- Reactive oxygen signaling and MAPK activation distinguish Epstein-Barr Virus (EBV)-positive versus EBV-negative Burkitt's lymphoma.
- Tumor necrosis factor-related apoptosis-inducing ligand can induce apoptosis in subsets of premalignant Cells.
- Involvement of p53 and p16 tumor suppressor genes in recessive dystrophic epidermolysis bullosa-associated Squamous cell carcinoma.
- Keloids demonstrate high-level epidermal expression of vascular endothelial growth factor.
- Molecular regulation of angiogenesis and tumorigenesis by signal transduction pathways: evidence of predictable and reproducible patterns of synergy in diverse neoplasms.
- Regulation of endothelial Cell differentiation and transformation by H-Ras.
- Activation of B-raf in non-malignant nevi predicts a novel tumor suppressor gene in Melanoma (MAP kinase phosphatase).
- Infectious angiogenesis: Bartonella bacilliformis Infection results in endothelial production of angiopoetin-2 and epidermal production of vascular endothelial growth ...
- Honokiol, a small molecular weight natural product, inhibits angiogenesis in vitro and tumor growth in vivo.
- Role of manganese superoxide dismutase in Cancer.
- Reversing the angiogenic switch with photodynamic therapy.
- Tuberous sclerosis-associated neoplasms express activated p42/44 mitogen-activated protein (MAP) kinase, and inhibition of MAP kinase signaling results in decreased in...
- Cutaneous Lesions of secondary syphilis are highly angiogenic.
- High-level expression of vascular endothelial growth factor and its receptors in an aphthous ulcer.
- Malignant transformation of melanocytes to Melanoma by constitutive activation of mitogen-activated protein kinase kinase (MAPKK) signaling.
- Molecular pathogenesis of vascular anomalies: classification into three categories based upon clinical and biochemical characteristics.
Schools
HARVARD MED SCH
Beth Israel Deaconess Hp M C
Mass Gen Hospital
Procedures Preformed
- Destruction of Benign Skin Lesion
- Destruction of Malignant Skin Lesion
Conditions Treated
- Actinic Keratosis
- Benign Skin Lesion
- Dermatitis
- Folliculitis
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Doctors Specialties
- Dermatology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Amerigroup (Wellpoint)
- Assurant Health
- AvMed
- Blue Card PPO
- Blue Choice HMO
- Blue Choice Option POS
- Blue Open Access POS
- Direct Access w/HSA
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- Coventry Health Care
- First Health (Coventry Health Care) PPO
- GEHA
- Choice Care PPO
- Preferred PPO
- Kaiser Permanente
- MedCost PPO
- Medical Mutual of Ohio
- Medicare MCR
- MultiPlan
- Mutual of Omaha
- Principal Life
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Education
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Massachusetts General Hospital -
Beth Israel Deaconess Medical Center -
Harvard Medical School
Hospital
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Emory University Hospital -
Children's Healthcare of Atlanta
Drug Facts
| NPI NUMBER | 1710092648 | |
| NPPES Provider LastName | ARBISER | |
| NPPES Provider FirstName | JACK | |
| NPPES Provider ZIPCode | 303221013 | |
| NPPES Provider State | GA | |
| Specialty Description | Dermatology | |
| Total Claim Count | 69.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1710092648 |
| Last Name Of The Provider | ARBISER |
| First Name Of The Provider | JACK |
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