Moseley Professor of Surgery, Emeritus, Brigham and Women's Hospital
Vascular Surgery
Dr. John A. Mannick, MD
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John Mannick practices Vascular Surgery. He went to medical school at Harvard Medical School. Harvard Medical School is a top medical college. Harvard Medical School has a rank of 1 in research and a 12 in primary care He received 2 awards: "Elected Member" and "Fellow (FACS)". John Mannick is a published doctor as well. He has 30 publications published. The latest was: Injury-Induced GR-1+ Macrophage Expansion and Activation Occurs Independent of CD4 T Cell Influence.
Publications
- Injury-Induced GR-1+ Macrophage Expansion and Activation Occurs Independent of CD4 T Cell Influence.
- Regulatory T cells suppress antigen-driven CD4 T cell reactivity following injury.
- Murine dendritic cell antigen-presenting cell function is not altered by burn injury.
- Twentieth anniversary of American Vascular Association/Lifeline Foundation: a celebration.
- Injury enhances resistance to Escherichia coli infection by boosting innate immune system function.
- Reviving the vascular surgeon-scientist: an interim assessment of the jointly sponsored Lifeline Foundation/National Heart, Lung, and Blood Institute William J. von Li...
- Increased CD4+ CD25+ T regulatory cell activity in trauma patients depresses protective Th1 immunity.
- Platelet depletion in mice increases mortality after thermal injury.
- Commonality and differences in leukocyte gene expression patterns among three models of inflammation and injury.
- Burn injury induces an early activation response by lymph node CD4+ T cells.
- Enhanced regulatory T cell activity is an element of the host response to injury.
- Enhanced TLR4 reactivity following injury is mediated by increased p38 activation.
- CD4+CD25+ regulatory T cells control innate immune reactivity after injury.
- Linking the "two-hit" response following injury to enhanced TLR4 reactivity.
- Thoracovisceral segment aneurysm repair after previous infrarenal abdominal aortic aneurysm surgery.
- Burn injury initiates a shift in superantigen-induced T cell responses and host survival.
- Injury, sepsis, and the regulation of Toll-like receptor responses.
- Burn injury promotes antigen-driven Th2-type responses in vivo.
- Use of intracellular cytokine staining and bacterial superantigen to document suppression of the adaptive immune system in injured patients.
- Interaction between the innate and adaptive immune systems is required to survive sepsis and control inflammation after injury.
- Injury primes the innate immune system for enhanced Toll-like receptor reactivity.
- Preoperative risk factors for carotid endarterectomy: defining the patient at high risk.
- The impact of patient age and aortic size on the results of aortobifemoral bypass grafting.
- Mast cells and resistance to peritoneal sepsis after burn injury.
- Outcome in patients at high risk after open surgical repair of abdominal aortic aneurysm.
- Bypass in the absence of ipsilateral greater saphenous vein: safety and superiority of the contralateral greater saphenous vein.
- Circadian rhythm of cytokine secretion following thermal injury in mice: implications for burn and trauma research.
- Usefulness of autogenous bypass grafts originating distal to the groin.
- 15th Annual Semmelweis Lecture Surgical Infection Society-Europe. Injury-induced immune dysfunction: is the lymphocyte irrelevant?
Schools
Harvard Medical School
Mass Genl Hospital
Doctors Specialties
- General Surgery
- Vascular Surgery
Accepted Insurances
Awards
- Elected Member
- Fellow (FACS)
Education
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Massachusetts General Hospital -
Harvard Medical School
Hospital
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Brigham and Women's Hospital
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