Physician
Pathology
Dr. Leo T. Furcht, MD
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Leo T. Furcht, MD practices Pathology. He attended State University of New York Upstate Medical University. Leo T. Furcht, MD is a published doctor and has 101 publications published. The lastest was: 'Effect of vinblastine sulfate, colchicine and lumicolchicine on membrane organization of normal and transformed cells.'
Publications
- Paradoxical roles for antioxidants in tumor prevention and eradication.
- NIH funding: what does the future look like?
- Financial disclosure policies of scientific publications.
- The potential role of neutrophils in promoting the metastatic phenotype of tumors releasing interleukin-8.
- Membrane type-1 matrix metalloproteinase promotes human melanoma invasion and growth.
- Atypical methylation of the interleukin-8 gene correlates strongly with the metastatic potential of breast carcinoma cells.
- Manipulation of hyaluronan synthase expression in prostate adenocarcinoma cells alters pericellular matrix retention and adhesion to bone marrow endothelial cells.
- Renal localization of heparan sulfate proteoglycan by immunohistochemistry.
- Production and characterization of monoclonal antibodies against avian retrovirus reverse transcriptase.
- Conflicts of interest in biomedical research--the FASEB guidelines.
- Localization of two unique heparin binding domains of human plasma fibronectin with monoclonal antibodies.
- Immunological identification of two sulfhydryl-containing fragments of human plasma fibronectin.
- Fibronectin involvement in granulation tissue and wound healing in rabbits.
- An axial periodic fibrillar arrangement of antigenic determinants for fibronectin and procollagen on ascorbate treated human fibroblasts.
- Effect of dibutyryl cyclic AMP and serum deprivation on the membranes of transformed mouse fibroblasts. A freeze fracture study.
- Influence of cell cycle and cell movement on the distribution of intramembranous particles in contact-inhibited and transformed cells.
- Differences in membrane fluidity and structure in contact-inhibited and transformed cells.
- Evidence that mitogenic lectins induce changes in lymphocyte membrane fluidity.
- Changes in membrane structure associated with cell contact.
- Alternative model for the internal structure of laminin.
- Extracellular matrix proteins (fibronectin, laminin, and type IV collagen) bind and aggregate bacteria.
- Location of a fibronectin domain involved in newt epidermal cell migration.
- Nonenzymatic glycation of fibronectin and alterations in the molecular association of cell matrix and basement membrane components in diabetes mellitus.
- Guidance of neurite outgrowth by pathways of substratum-adsorbed laminin.
- Neuron-specific interactions with two neurite-promoting fragments of fibronectin.
- Laminin selectively enhances monocyte-macrophage-mediated tumoricidal activity.
- Human fibronectin contains distinct adhesion- and motility-promoting domains for metastatic melanoma cells.
- The role of cell adhesion proteins--laminin and fibronectin--in the movement of malignant and metastatic cells.
- Clearing and release of basement membrane proteins from substrates by metastatic tumor cell variants.
- Localization of a tumor cell adhesion domain of laminin by a monoclonal antibody.
- Decreased interaction of fibronectin, type IV collagen, and heparin due to nonenzymatic glycation. Implications for diabetes mellitus.
- A novel synthetic peptide from the B1 chain of laminin with heparin-binding and cell adhesion-promoting activities.
- Transient expression of laminin in the optic nerve of the developing rat.
- Differential effects of laminin, intact type IV collagen, and specific domains of type IV collagen on endothelial cell adhesion and migration.
- Molecular mechanisms in basement membrane complications of diabetes. Alterations in heparin, laminin, and type IV collagen association.
- Effects of matrix proteins on rabbit corneal epithelial cell adhesion and migration.
- Localization of three distinct heparin-binding domains of laminin by monoclonal antibodies.
- The effect of nonenzymatic glucosylation on the binding of the main noncollagenous NC1 domain to type IV collagen.
- Metastasis inhibition of different tumor types by purified laminin fragments and a heparin-binding fragment of fibronectin.
- Growth cone guidance by substrate-bound laminin pathways is correlated with neuron-to-pathway adhesivity.
- Immunoreactivity for laminin in the developing ventral longitudinal pathway of the brain.
- Epithelial and neural localization and heparin binding of the cell-substratum adhesion molecule, epinectin.
- Growth cone migration across extracellular matrix components depends on integrin, but migration across glioma cells does not.
- C1q component of complement binds to fibrinogen and fibrin.
- Cell adhesion and neurite extension in response to two proteolytic fragments of laminin.
- Heparin type IV collagen interactions: equilibrium binding and inhibition of type IV collagen self-assembly.
- Localization and chemical synthesis of fibronectin peptides with melanoma adhesion and heparin binding activities.
- Selective interaction of peripheral and central nervous system cells with two distinct cell-binding domains of fibronectin.
- The binding of heparin to type IV collagen: domain specificity with identification of peptide sequences from the alpha 1(IV) and alpha 2(IV) which preferentially bind ...
- Mapping of three major heparin-binding sites on laminin and identification of a novel heparin-binding site on the B1 chain.
- Quantification of corneal organ culture migration: central and peripheral epithelium.
- Immunohistochemical localization of fibronectin in the chinchilla cochlea.
- Novel synthetic heparin binding peptides of laminin and fibronectin which promote the adhesion of melanoma cells.
- Immunohistochemical localization of laminin in the basement membranes of normal, hyperplastic, and neoplastic human prostate.
- Transforming growth factor-beta 1 binds to immobilized fibronectin.
- Stimulation of haptotaxis and migration of tumor cells by serum spreading factor.
- Granulocyte-mediated injury to herpes simplex virus-infected human endothelium.
- Herpes simplex virus inhibits endothelial cell attachment and migration to extracellular matrix proteins.
- Adhesion of lymphoid cells to the carboxyl-terminal heparin-binding domains of fibronectin.
- G-protein involvement in matrix-mediated motility and invasion of high and low experimental metastatic B16 melanoma clones.
- Critical factors controlling angiogenesis: cell products, cell matrix, and growth factors.
- Human keratinocytes adhere to a unique heparin-binding peptide sequence within the triple helical region of type IV collagen.
- Characterization of a synthetic peptide from type IV collagen that promotes melanoma cell adhesion, spreading, and motility.
- RGD-independent cell adhesion to the carboxy-terminal heparin-binding fragment of fibronectin involves heparin-dependent and -independent activities.
- Differential effects of transforming growth factor beta 1 on the growth of poorly and highly metastatic murine melanoma cells.
- Recognition of the A chain carboxy-terminal heparin binding region of fibronectin involves multiple sites: two contiguous sequences act independently to promote neural...
- Definition of a sequence, RYVVLPR, within laminin peptide F-9 that mediates metastatic fibrosarcoma cell adhesion and spreading.
- Identification of a multifunctional, cell-binding peptide sequence from the a1(NC1) of type IV collagen.
- A heparan sulfate-containing fraction of bone marrow stroma induces maturation of HL-60 cells in vitro.
- The effect of transforming growth factor-beta 1 on glycosaminoglycan production by human marrow cultures.
- Laminin alterations after in vitro nonenzymatic glycosylation.
- An overview of role of matrix components.
- Synthetic peptides from the carboxy-terminal globular domain of the A chain of laminin: their ability to promote cell adhesion and neurite outgrowth, and interact with...
- 1H NMR and CD secondary structure analysis of cell adhesion promoting peptide F-9 from laminin.
- Tumor cell adhesive mechanisms and their relationship to metastasis.
- Type IV collagen and corneal epithelial adhesion and migration. Effects of type IV collagen fragments and synthetic peptides on rabbit corneal epithelial cell adhesion...
- Human keratinocytes adhere to two distinct heparin-binding synthetic peptides derived from fibronectin.
- The role of G-protein in matrix-mediated motility of highly and poorly invasive melanoma cells.
- Effects of laminin on functional reinnervation of target organs by regenerating axons.
- Localization of type IV collagen in the basement membranes of human prostate and lymph nodes by immunoperoxidase and immunoalkaline phosphatase.
- Coordinate role for cell surface chondroitin sulfate proteoglycan and alpha 4 beta 1 integrin in mediating melanoma cell adhesion to fibronectin.
- A cell-surface heparan sulfate proteoglycan mediates neural cell adhesion and spreading on a defined sequence from the C-terminal cell and heparin binding domain of fi...
- Central and peripheral neurite outgrowth differs in preference for heparin-binding versus integrin-binding sequences.
- Cell surface phosphatidylinositol-anchored heparan sulfate proteoglycan initiates mouse melanoma cell adhesion to a fibronectin-derived, heparin-binding synthetic pept...
- A synthetic peptide derived from the carboxy terminus of the laminin A chain represents a binding site for the alpha 3 beta 1 integrin.
- Effects of transforming growth factor-beta 1 on human pulmonary adenocarcinoma cell adhesion, motility, and invasion in vitro.
- The emerging role of molecular diagnostics in surgical pathology.
- Effect of glow discharge surface modification of plasma TFE vascular graft material on fibronectin and laminin retention and endothelial cell adhesion.
- Rabbit corneal epithelial cells adhere to two distinct heparin-binding synthetic peptides derived from fibronectin.
- Effect of vinblastine sulfate, colchicine and lumicolchicine on membrane organization of normal and transformed cells.
- Membrane pathology of normal and malignant cells--a review.
- Effects of cell density and transformation on the formation of a fibronectin extracellular filamentous matrix on human fibroblasts.
- Cell surface changes accompanying myoblast differentiation.
- Trypsin-induced coordinate alterations in cell shape, cytoskeleton, and intrinsic membrane structure of contact-inhibited cells.
- Plasma membrane intramembranous particle topography in 3T3 and SV3T3 cells: the effect of cytochalasin B.
- Immunocytochemical localization of fibronectin (LETS proteins) on the surface of L6 myoblasts: light and electron microscopic studies.
- Dexamethasone-induced accumulation of a fibronectin and collagen extracellular matrix in transformed human cells.
- Differences in the ultrastructural localization of fibronectin on normal and transformed cells.
- Reversal by glucocorticoid hormones of the loss of a fibronectin and probollagen matrix around transformed human cells.
- Changes in intramembranous particle topography and concanavalin A receptor mobility associated with myoblast differentiation.
Schools
SUNY Downstate Medical Center College Of Medicine
Fairview University Med Center
Doctors Specialties
- Clinical Pathology
- Pathology
Accepted Insurances
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Choice Plus POS
- Navigate POS
- Options PPO
Education
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University of Minnesota at Fairview -
State University of New York Upstate Medical University
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