Physician
Internal Medicine
Dr. Luis S. Ulloa, MD
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Doctor Luis S. Ulloa, MD works in the field of Internal Medicine. He went to medical school at New York Medical College. He over the years received 1 awards: "Meaningful Use Stage 1 Certification". Luis Ulloa is also a published doctor. He has 64 publication published. The most recent publication is: Interactions between cellular actin and human respiratory syncytial virus (HRSV). He is registered with Medicare and accepts Medicare payments.
Publications
- Parasympathetic stimulation via the vagus nerve prevents systemic organ dysfunction by abrogating gut injury and lymph toxicity in trauma and hemorrhagic shock.
- Lefty inhibits receptor-regulated Smad phosphorylation induced by the activated transforming growth factor-beta receptor.
- Lefty proteins exhibit unique processing and activate the MAPK pathway.
- Inhibition of transforming growth factor-beta/SMAD signalling by the interferon-gamma/STAT pathway.
- Interactions between cellular actin and human respiratory syncytial virus (HRSV).
- The phosphorylated isoform of microtubule associated protein 1B (MAP1B) is expressed in the visual system of the tench (Tinca tinca, L) during optic nerve regeneration.
- NMDA-glutamate receptors regulate phosphorylation of dendritic cytoskeletal proteins in the hippocampus.
- Phosphorylated microtubule associated protein (MAP1B-Phos) in the retina during regeneration of the fish optic nerve.
- Analysis of the expression, distribution and function of cyclin dependent kinase 5 (cdk5) in developing cerebellar macroneurons.
- Involvement of gamma and beta actin isoforms in mouse neuroblastoma differentiation.
- Characterization of microtubule-associated protein MAP1B: phosphorylation state, light chains, and binding to microtubules.
- Depletion of casein kinase II by antisense oligonucleotide prevents neuritogenesis in neuroblastoma cells.
- Localization of differentially phosphorylated isoforms of microtubule-associated protein 1B in cultured rat hippocampal neurons.
- Microtubule-associated protein MAP1B showing a fetal phosphorylation pattern is present in sites of neurofibrillary degeneration in brains of Alzheimer''s disease pati...
- Dephosphorylation of distinct sites on microtubule-associated protein MAP1B by protein phosphatases 1, 2A and 2B.
- Heterogeneity in the phosphorylation of microtubule-associated protein MAP1B during rat brain development.
- Depletion of catalytic and regulatory subunits of protein kinase CK2 by antisense oligonucleotide treatment of neuroblastoma cells.
- Phosphorylation of microtubule-associated proteins by protein kinase CK2 in neuritogenesis.
- Microtubule-associated protein 1B (MAP1B) is present in glial cells phosphorylated different than in neurones.
- Role of phosphorylated MAPlB in neuritogenesis.
- Chemoimmunotherapy versus chemotherapy for metastatic malignant melanoma.
- High mobility group box chromosomal protein 1: a novel proinflammatory mediator in synovitis.
- The alpha7 nicotinic acetylcholine receptor as a pharmacological target for inflammation.
- Antioxidants preserve macrophage phagocytosis of Pseudomonas aeruginosa during hyperoxia.
- The neuronal strategy for inflammation.
- Ethyl pyruvate prevents lethality in mice with established lethal sepsis and systemic inflammation.
- Globin attenuates the innate immune response to endotoxin.
- Pharmacological stimulation of the cholinergic antiinflammatory pathway.
- ?2-Adrenoreceptors of regulatory lymphocytes are essential for vagal neuromodulation of the innate immune system.
- The anti-inflammatory potential of selective cholinergic agonists.
- Cholinergic regulatory lymphocytes re-establish neuromodulation of innate immune responses in sepsis.
- ?7-cholinergic receptor mediates vagal induction of splenic norepinephrine.
- Unphosphorylated STAT3 modulates alpha 7 nicotinic receptor signaling and cytokine production in sepsis.
- Novel insights for systemic inflammation in sepsis and hemorrhage.
- Fat and the gut: more than empty calories.
- Mast cell stabilization improves survival by preventing apoptosis in sepsis.
- Oral activated charcoal prevents experimental cerebral malaria in mice and in a randomized controlled clinical trial in man did not interfere with the pharmacokinetics...
- JAK2 inhibition prevents innate immune responses and rescues animals from sepsis.
- Survival and inflammatory responses in experimental models of hemorrhage.
- Ethyl pyruvate prevents inflammatory responses and organ damage during resuscitation in porcine hemorrhage.
- Alpha7 cholinergic-agonist prevents systemic inflammation and improves survival during resuscitation.
- Scientific and clinical challenges in sepsis.
- Anti-inflammatory resuscitation improves survival in hemorrhage with trauma.
- Neuroimmune perspectives in sepsis.
- Ethyl pyruvate improves survival in awake hemorrhage.
- Alpha7 nicotinic acetylcholine receptor: a link between inflammation and neurodegeneration.
- High-mobility group box-1 isoforms as potential therapeutic targets in sepsis.
- Splenectomy inactivates the cholinergic antiinflammatory pathway during lethal endotoxemia and polymicrobial sepsis.
- The vagus nerve and nicotinic receptors modulate experimental pancreatitis severity in mice.
- High-mobility group box 1 (HMGB1) protein: friend and foe.
- Hmgb-1 as a therapeutic target for infectious and inflammatory disorders.
- The vagus nerve and the nicotinic anti-inflammatory pathway.
- Alpha-chemokine receptor blockade reduces high mobility group box 1 protein-induced lung inflammation and injury and improves survival in sepsis.
- Adrenomedullin and its binding protein attenuate the proinflammatory response after hemorrhage.
- The "cytokine profile": a code for sepsis.
- Suppression of HMGB1 release by stearoyl lysophosphatidylcholine:an additional mechanism for its therapeutic effects in experimental sepsis.
- Cholinergic agonists inhibit HMGB1 release and improve survival in experimental sepsis.
- Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14- and TNF-dependent mechanisms.
- Recombinant HMGB1 with cytokine-stimulating activity.
- Reversing established sepsis with antagonists of endogenous high-mobility group box 1.
- Structural basis for the proinflammatory cytokine activity of high mobility group box 1.
- High mobility group box chromosomal protein 1 as a nuclear protein, cytokine, and potential therapeutic target in arthritis.
- Nicotinic acetylcholine receptor alpha7 subunit is an essential regulator of inflammation.
Schools
New York Medical College
Doctors Specialties
- Internal Medicine
Accepted Insurances
Awards
- Meaningful Use Stage 1 Certification
Education
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New York Medical College
Drug Facts
| NPI NUMBER | 1457318636 | |
| NPPES Provider LastName | ULLOA | |
| NPPES Provider FirstName | LUIS | |
| NPPES Provider ZIPCode | 334582788 | |
| NPPES Provider State | FL | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 3830.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1457318636 |
| Last Name Of The Provider | ULLOA |
| First Name Of The Provider | LUIS |
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