Professor of Neurology, Washington University School of Medicine
Neurology
Dr. Marc I. Diamond, MD
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Marc I. Diamond is an expert in the field of Neurology. He went to medical school at University of California San Francisco School of Medicine. University of California San Francisco School of Medicine is a one of the best medicals schools. University of California San Francisco School of Medicine is ranked 3/3 in Research/PrimaryCare. Marc I. Diamond is a published physician. He published 47 publications, including: 'Prions and Protein Assemblies that Convey Biological Information in Health and Disease.'
Publications
- Prions and Protein Assemblies that Convey Biological Information in Health and Disease.
- Propagation of prions causing synucleinopathies in cultured cells.
- Anti-tau antibody reduces insoluble tau and decreases brain atrophy.
- Subcellular Localization and Ser-137 Phosphorylation Regulate Tumor-Suppressive Activity of Profilin-1.
- N17 Modifies Mutant Huntingtin Nuclear Pathogenesis and Severity of Disease in HD BAC Transgenic Mice.
- The green tea polyphenol (-)-epigallocatechin gallate prevents the aggregation of tau protein into toxic oligomers at substoichiometric ratios.
- Inhibition of the androgen receptor by mineralocorticoids at levels physiologically achieved in serum in patients treated with abiraterone acetate.
- Distinct tau prion strains propagate in cells and mice and define different tauopathies.
- Prion-like Properties of tau protein: The importance of extracellular tau as a therapeutic target.
- Anti-Tau Antibodies that Block Tau Aggregate Seeding In Vitro Markedly Decrease Pathology and Improve Cognition In Vivo.
- Heparan sulfate proteoglycans mediate internalization and propagation of specific proteopathic seeds.
- Prion-like nuclear aggregation of TDP-43 during heat shock is regulated by HSP40/70 chaperones.
- Prion-like propagation of protein aggregation and related therapeutic strategies.
- Mechanisms of protein seeding in neurodegenerative Diseases.
- Intravitreal Administration of HA-1077, a ROCK Inhibitor, Improves Retinal Function in a Mouse Model of Huntington Disease.
- An N-terminal Nuclear Export Signal Regulates Trafficking and Aggregation of Htt exon 1.
- Amyotrophic lateral sclerosis and organ donation: is there risk of Disease transmission?
- An aggregation sensing reporter identifies leflunomide and teriflunomide as polyglutamine aggregate inhibitors.
- In Vivo Microdialysis Reveals Age-Dependent Decrease of Brain Interstitial Fluid Tau Levels in P301S Human Tau Transgenic Mice.
- Interaction with polyglutamine aggregates reveals a Q/N-rich domain in TDP-43.
- F-actin binding regions on the androgen receptor and huntingtin increase aggregation and alter aggregate characteristics.
- Y-27632 improves rotarod performance and reduces huntingtin levels in R6/2 mice.
- Propagation of tau misfolding from the outside to the inside of a cell.
- Non-competitive androgen receptor inhibition in vitro and in vivo.
- The expanding realm of prion phenomena in neurodegenerative Disease.
- Discovery of selective glucocorticoid receptor modulators by multiplexed reporter screening.
- AR inhibitors identified by high-throughput microscopy detection of conformational change and subcellular localization.
- Conformational diversity of wild-type Tau fibrils specified by templated conformation change.
- Phosphorylation of profilin by ROCK1 regulates polyglutamine aggregation.
- A common motif targets huntingtin and the androgen receptor to the proteasome.
- A cellular conformation-based screen for androgen receptor inhibitors.
- Design and implementation of cell-based assays to model human Disease.
- Soluble androgen receptor oligomers underlie pathology in a mouse model of spinobulbar muscular atrophy.
- Selective activation of estrogen receptor-beta transcriptional pathways by an herbal extract.
- The structural basis of androgen receptor activation: intramolecular and intermolecular amino-carboxy interactions.
- A rapid cellular FRET assay of polyglutamine aggregation identifies a novel inhibitor.
- Advances in diagnostic testing for Alzheimer Disease.
- Joints in the regulatory lattice: composite regulation by steroid receptor-AP1 complexes.
- Transcription factor interactions: selectors of positive or negative regulation from a single DNA element.
- Regulation of expanded polyglutamine protein aggregation and nuclear localization by the glucocorticoid receptor.
- Polyglutamine protein aggregation and toxicity are linked to the cellular stress response.
- Glucocorticoid modulation of androgen receptor nuclear aggregation and cellular toxicity is associated with distinct forms of soluble expanded polyglutamine protein.
- Biologically active molecules that reduce polyglutamine aggregation and toxicity.
- Polyglutamine Diseases: emerging concepts in pathogenesis and therapy.
- Prion-like mechanisms in neurodegenerative Diseases.
- ROCK and PRK-2 mediate the inhibitory effect of Y-27632 on polyglutamine aggregation.
Schools
University of California At San Francisco
University Ca Sf Med Center
University Ca Sf School Of Med
Conditions Treated
- Ataxia
- Cerebral Artery Thrombosis
- Chorea (Excluding Huntington's Chorea)
- Convulsions
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Doctors Specialties
- Neurology
Accepted Insurances
Education
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UCSF -
University of California San Francisco School of Medicine
Hospital
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St. Louis Children's Hospital
Drug Facts
| NPI NUMBER | 1255368288 | |
| NPPES Provider LastName | DIAMOND | |
| NPPES Provider FirstName | MARC | |
| NPPES Provider ZIPCode | 631101032 | |
| NPPES Provider State | MO | |
| Specialty Description | Neurology | |
| Total Claim Count | 208.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1255368288 |
| Last Name Of The Provider | DIAMOND |
| First Name Of The Provider | MARC |
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