Professor, Internal Medicine, University of Kentucky College of Medicine
Endocrinology
Dr. Philip A. Kern, MD
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Dr. Philip A. Kern practices Endocrinology. He graduated from New York Medical College. He has 3 awards "Regional Top Doctor", "CMS Meaningful Use Stage 1 Certification" and "Elected Member". Philip A. Kern, MD is a published doctor as well. He has 58 publications published. The latest was: IKK is essential for adipocyte survival and adaptive adipose remodeling in obesity. Dr. Philip A. Kern is registered with Medicare.
Publications
- Integrative mRNA-microRNA analyses reveal novel interactions related to insulin sensitivity in human adipose tissue.
- The triglyceride to high-density lipoprotein cholesterol (TG/HDL-C) ratio as a predictor of β-cell function in African American women.
- Increasing Adipocyte Lipoprotein Lipase Improves Glucose Metabolism in High Fat Diet Induced Obesity.
- Social Network Analysis to Assess the Impact of the CTSA on Biomedical Research Grant Collaboration.
- The Effects of Temperature and Seasons on Subcutaneous White Adipose Tissue in Humans: Evidence for Thermogenic Gene Induction.
- Fiber Type-specific satellite cell response to aerobic training in sedentary adults.
- An ACACB Variant Implicated in Diabetic Nephropathy Associates with Body Mass Index and Gene Expression in Obese Subjects.
- Regulation of thrombospondin-1 expression in alternatively activated macrophages and adipocytes: role of cellular cross talk and omega-3 fatty acids.
- Omega-3 Fatty Acids Reduce Adipose Tissue Macrophages in Human Subjects With Insulin Resistance.
- Regulation of Small Ubiquitin-Like Modifier-1, Nuclear Receptor Coreceptor, Histone Deacetylase 3, and Peroxisome Proliferator-Activated Receptor-γ in Human Adipose Ti...
- Improved insulin sensitivity after treatment with PPARγ and PPARα ligands is mediated by genetically modulated transcripts.
- The lipoprotein lipase (LPL) S447X gain of function variant involves increased mRNA translation.
- Adipose tissue extracellular matrix and vascular Abnormalities in obesity and insulin resistance.
- DHA reduces the atrophy-associated Fn14 protein in differentiated myotubes during coculture with macrophages.
- Obesity and hepatosteatosis in mice with enhanced oxidative DNA damage processing in mitochondria.
- Global gene expression profiles of subcutaneous adipose and muscle from glucose-tolerant, insulin-sensitive, and insulin-resistant individuals matched for BMI.
- Adipose triglyceride lipase expression in human adipose tissue and muscle. Role in insulin resistance and response to training and pioglitazone.
- Adipose tissue macrophages in insulin-resistant subjects are associated with collagen VI and fibrosis and demonstrate alternative activation.
- Insulin resistance in African-American and Caucasian women: differences in lipotoxicity, adipokines, and gene expression in adipose tissue and muscle.
- Matrix metalloproteinase-9 is increased in obese subjects and decreases in response to pioglitazone.
- The impact of exercise training compared to caloric restriction on hepatic and peripheral insulin resistance in obesity.
- Association of scavenger receptors in adipose tissue with insulin resistance in nondiabetic humans.
- Muscle inflammatory response and insulin resistance: synergistic interaction between macrophages and fatty acids leads to impaired insulin action.
- Adiponectin translation is increased by the PPARgamma agonists pioglitazone and omega-3 fatty acids.
- Visceral fat and adiponectin: associations with insulin resistance are tissue-specific in women.
- Adipocytokines and the metabolic complications of obesity.
- Stearoyl-coenzyme A desaturase 1 gene expression increases after pioglitazone treatment and is associated with peroxisomal proliferator-activated receptor-gamma respon...
- Endoplasmic reticulum stress markers are associated with obesity in nondiabetic subjects.
- Effect of pioglitazone treatment on endoplasmic reticulum stress response in human adipose and in palmitate-induced stress in human liver and adipose cell lines.
- Calcium is involved in formation of high molecular weight adiponectin.
- Thrombospondin-1 is an adipokine associated with obesity, adipose inflammation, and insulin resistance.
- Retinol binding protein 4 expression in humans: relationship to insulin resistance, inflammation, and response to pioglitazone.
- Human visfatin expression: relationship to insulin sensitivity, intramyocellular Lipids, and inflammation.
- Effects of pioglitazone and metformin on beta-cell function in nondiabetic subjects at high risk for type 2 Diabetes.
- OXPAT/PAT-1 is a PPAR-induced Lipid droplet protein that promotes fatty acid utilization.
- Pioglitazone increases secretion of high-molecular-weight adiponectin from adipocytes.
- The lipogenic enzymes DGAT1, FAS, and LPL in adipose tissue: effects of obesity, insulin resistance, and TZD treatment.
- Lipin expression is attenuated in adipose tissue of insulin-resistant human subjects and increases with peroxisome proliferator-activated receptor gamma activation.
- The prevention and treatment of metabolic Syndrome and high-risk obesity.
- Pioglitazone induces apoptosis of macrophages in human adipose tissue.
- Increased plasma adiponectin in response to pioglitazone does not result from increased gene expression.
- Role of A kinase anchor proteins in the tissue-specific regulation of lipoprotein lipase.
- Expression of CD68 and macrophage chemoattractant protein-1 genes in human adipose and muscle tissues: association with cytokine expression, insulin resistance, and re...
- Pioglitazone improves insulin sensitivity through reduction in muscle Lipid and redistribution of Lipid into adipose tissue.
- Divergent effects of peroxisome proliferator-activated receptor gamma agonists and tumor necrosis factor alpha on adipocyte ApoE expression.
- Expression of FOXC2 in adipose and muscle and its association with whole body insulin sensitivity.
- Insulin sensitivity following agent orange exposure in Vietnam veterans with high blood levels of 2,3,7,8-tetrachlorodibenzo-p-dioxin.
- Lipid and carbohydrate Metabolism in mice with a targeted mutation in the IL-6 gene: absence of development of age-related obesity.
- Perilipin expression in human adipose tissue is elevated with obesity.
- Uncoupling protein-2 polymorphisms in type 2 Diabetes, obesity, and insulin secretion.
- Transgenic mice expressing lipoprotein lipase in adipose tissue. Absence of the proximal 3'-untranslated region causes translational upregulation.
- Adiponectin expression from human adipose tissue: relation to obesity, insulin resistance, and tumor necrosis factor-alpha expression.
- The translational regulation of lipoprotein lipase by epinephrine involves an RNA binding complex including the catalytic subunit of protein kinase A.
- Regulation of lipoprotein lipase by protein kinase C alpha in 3T3-F442A adipocytes.
- The stimulation of tumor necrosis factor and inhibition of glucose transport and lipoprotein lipase in adipose cells by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
- Pioglitazone Treatment Reduces Adipose Tissue Inflammation through Reduction of Mast Cell and Macrophage Number and by Improving Vascularity.
- IKKβ is essential for adipocyte survival and adaptive adipose remodeling in obesity.
Schools
New York Medical College
University Colo Health Scis Center
Conditions Treated
- Abnormal Thyroid
- Acromegaly and Gigantism
- Calcium Metabolism Disorders
- Diabetes Type 2
- View All
Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
- Elected Member
Education
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University of Colorado -
Albert Einstein - Montefiore Medical Center -
New York Medical College
Hospital
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University of Kentucky Albert B. Chandler Hospital
Drug Facts
| NPI NUMBER | 1114034170 | |
| NPPES Provider LastName | KERN | |
| NPPES Provider FirstName | PHILIP | |
| NPPES Provider ZIPCode | 405360298 | |
| NPPES Provider State | KY | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 98.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1114034170 |
| Last Name Of The Provider | KERN |
| First Name Of The Provider | PHILIP |
| View All |
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