Professor of Ophthalmology, Boston Children's Hospital
Ophthalmology
Dr. Robert J. D'Amato, MD
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Robert J. D'Amato practices Ophthalmology. He went to Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is one of the top medical schools in the US. Johns Hopkins University School of Medicine is ranked 3/29 in Research/PrimaryCare. Robert D'Amato is a published doctor and has 62 publications published. The lastest was: 'Involvement of platelets in tumour angiogenesis?'
Publications
- Cellular mechanism of oral absorption of solidified polymer micelles.
- Melanocyte pigmentation inversely correlates with MCP-1 production and angiogenesis-inducing potential.
- Pomalidomide is strongly antiangiogenic and teratogenic in relevant animal models.
- The stem cell marker prominin-1/CD133 interacts with vascular endothelial growth factor and potentiates its action.
- The albino mutation of tyrosinase alters ocular angiogenic responsiveness.
- The classical pink-eyed dilution mutation affects angiogenic responsiveness.
- Forty-year journey of angiogenesis translational research.
- A Morphometric Study of Mechanotransductively Induced Dermal Neovascularization.
- Green tea epigallocatechin-3-gallate inhibits angiogenesis and suppresses vascular endothelial growth factor C/vascular endothelial growth factor receptor 2 expression...
- Circulating endothelial progenitor cells are up-regulated in a mouse model of endometriosis.
- Angiogenesis in wounds treated by microdeformational wound therapy.
- Broad spectrum antiangiogenic treatment for ocular neovascular diseases.
- Genetic loci that control the size of laser-induced choroidal neovascularization.
- Differential suppression of vascular permeability and corneal angiogenesis by nonsteroidal anti-inflammatory drugs.
- Dendritic cells augment choroidal neovascularization.
- An orally delivered small-molecule formulation with antiangiogenic and anticancer activity.
- Dendritic cells support angiogenesis and promote lesion growth in a murine model of endometriosis.
- 2-methoxyestradiol inhibits hypoxia-inducible factor-1{alpha} and suppresses growth of lesions in a mouse model of endometriosis.
- Mutant anthrax toxin B moiety (protective antigen) inhibits angiogenesis and tumor growth.
- Angiogenesis and antiangiogenic therapy in endometriosis.
- Chronic suppression of angiogenesis following radiation exposure is independent of hematopoietic reconstitution.
- A novel noninvasive model of endometriosis for monitoring the efficacy of antiangiogenic therapy.
- The effect of genetic diversity on angiogenesis.
- Analysis of tumor-associated stromal cells using SCID GFP transgenic mice: contribution of local and bone marrow-derived host cells.
- Short synthetic endostatin peptides inhibit endothelial migration in vitro and endometriosis in a mouse model.
- Endostatin inhibits the growth of endometriotic lesions but does not affect fertility.
- X-linked dominant growth suppression of transplanted tumors in C57BL/6J-scid mice.
- Genetic heterogeneity of the vasculogenic phenotype parallels angiogenesis; Implications for cellular surrogate marker analysis of antiangiogenesis.
- Nonsteroidal antiinflammatory drugs differentially suppress endometriosis in a murine model.
- Genetic loci that control the angiogenic response to basic fibroblast growth factor.
- Genetic loci that control vascular endothelial growth factor-induced angiogenesis.
- Persistence of microscopic human cancers in mice: alterations in the angiogenic balance accompanies loss of tumor dormancy.
- S-3-Amino-phthalimido-glutarimide inhibits angiogenesis and growth of B-cell neoplasias in mice.
- Injection of antiangiogenic agents into the macaque preovulatory follicle: disruption of corpus luteum development and function.
- Characterization of a spontaneous retinal neovascular mouse model.
- Interactions of 2-methoxyestradiol, an endogenous mammalian metabolite, with unpolymerized tubulin and with tubulin polymers.
- Angiogenesis inhibition in age-related macular degeneration.
- Strain-dependent anterior segment neovascularization following intravitreal gene transfer of basic fibroblast growth factor (bFGF).
- Involvement of platelets in tumour angiogenesis?
- Treatment of the Kasabach-Merritt syndrome with pegylated recombinant human megakaryocyte growth and development factor in mice: elevated platelet counts, prolonged su...
- New activity of spironolactone. Inhibition of angiogenesis in vitro and in vivo.
- Thalidomide is an inhibitor of angiogenesis.
- 2-Methoxyestradiol, an endogenous mammalian metabolite, inhibits tubulin polymerization by interacting at the colchicine site.
- Effects of thalidomide and related metabolites in a mouse corneal model of neovascularization.
- Exogenous control of mammalian gene expression through modulation of RNA self-cleavage.
- Regulation of vascular endothelial growth factor expression by insulin-like growth factor I.
- The antiangiogenic agents TNP-470 and 2-methoxyestradiol inhibit the growth of angiosarcoma in mice.
- Critical components of the female reproductive pathway are suppressed by the angiogenesis inhibitor AGM-1470.
- The mouse cornea micropocket angiogenesis assay.
- Mechanism of action of thalidomide and 3-aminothalidomide in multiple myeloma.
- Inhibition of angiogenesis and breast cancer in mice by the microtubule inhibitors 2-methoxyestradiol and taxol.
- Rapid ocular angiogenic control via naked DNA delivery to cornea.
- Combination oral antiangiogenic therapy with thalidomide and sulindac inhibits tumour growth in rabbits.
- Experimental corneal neovascularisation using sucralfate and basic fibroblast growth factor.
- Inhibition of angiogenesis in vivo by interleukin 12.
- Matrigel cytometry: a novel method for quantifying angiogenesis in vivo.
- Antiangiogenic effect of oral 2-methoxyestradiol on choroidal neovascularization in mice.
- Intrachoroidal neovascularization in transgenic mice overexpressing vascular endothelial growth factor in the retinal pigment epithelium.
- Cytochalasin E, an epoxide containing Aspergillus-derived fungal metabolite, inhibits angiogenesis and tumor growth.
- A model of angiogenesis in the mouse cornea.
- Genetic heterogeneity of angiogenesis in mice.
Schools
Johns Hopkins University School Of Medicine
Conditions Treated
- Glaucoma
- Pinkeye (Conjunctivitis)
Doctors Specialties
- Ophthalmology
Accepted Insurances
Education
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Massachusetts Eye & Ear Infirmary -
Johns Hopkins University School of Medicine
Hospital
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Boston Children's Hospital
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