Physician

Infectious Disease

Dr. Robert S. Munford III, MD

Robert Munford practices Infectious Disease. He went to Harvard Medical School. Harvard Medical School is a top medical university. Harvard Medical School is ranked 1/12 in Research/PrimaryCare. Robert Munford is a published doctor. He published 25 publications, including: 'Prolonged Triglyceride Storage in Macrophages: pHo Trumps pO2 and TLR4.'

Publications

Prolonged Triglyceride Storage in Macrophages: pHo Trumps pO2 and TLR4.
Persistently active microbial molecules prolong innate immune tolerance in vivo.
Hepatic uptake and deacylation of the LPS in bloodborne LPS-lipoprotein complexes.
The transport and inactivation kinetics of bacterial lipopolysaccharide influence its immunological potency in vivo.
Novel therapies for septic shock over the past 4 decades.
Prolonged hepatomegaly in mice that cannot inactivate bacterial endotoxin.
Murine responses to endotoxin: another dirty little secret?
Overproduction of acyloxyacyl hydrolase by macrophages and dendritic cells prevents prolonged reactions to bacterial lipopolysaccharide in vivo.
Host inactivation of bacterial lipopolysaccharide prevents prolonged tolerance following gram-negative bacterial infection.
Sensing gram-negative bacterial lipopolysaccharides: a human disease determinant?
A host lipase detoxifies bacterial lipopolysaccharides in the liver and spleen.
Endotoxin-binding proteins modulate the susceptibility of bacterial endotoxin to deacylation by acyloxyacyl hydrolase.
Shield as signal: lipopolysaccharides and the evolution of immunity to gram-negative bacteria.
Severe sepsis and septic shock: the role of gram-negative bacteremia.
Lipopolysaccharide deacylation by an endogenous lipase controls innate antibody responses to Gram-negative bacteria.
Recurrent exposure to Histoplasma capsulatum in modern air-conditioned buildings.
Innate immunity genes influence the severity of acute appendicitis.
Identification of acyloxyacyl hydrolase, a lipopolysaccharide-detoxifying enzyme, in the murine urinary tract.
Acute inflammation and infection maintain circulating phospholipid levels and enhance lipopolysaccharide binding to plasma lipoproteins.
Stimulus-dependent deacylation of bacterial lipopolysaccharide by dendritic cells.
Regional and systemic cytokine responses to acute inflammation of the vermiform appendix.
Risks and benefits of activated protein C treatment for severe sepsis.
Is severe sepsis a neuroendocrine disease?
The G-->A single nucleotide polymorphism at the -308 position in the tumor necrosis factor-alpha promoter increases the risk for severe sepsis after trauma.

Schools

Harvard Medical School
Parkland Memorial Hospital
Massachusetts General Hospital

Doctors Specialties

Infectious Disease Medicine
Infectious Disease

Accepted Insurances

Education

Massachusetts General Hospital
Harvard Medical School

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