Physician
Pulmonology
Dr. Theodore J. Standiford, MD
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Theodore J. Standiford practices Pulmonology. He went to medical school at Wayne State University School of Medicine. Wayne State University School of Medicine is ranked 63 in research and 85 in primary care medicine He received 2 awards: "CMS Meaningful Use Stage 1 Certification" and "Elected Member". Theodore Standiford is a published doctor. He published 83 publications, including: 'Cecal Ligation and Puncture Results in Long-Term Central Nervous System Myeloid Inflammation.' He is registered with Medicare and accepts Medicare payments.
Publications
- Micro RNA 155 regulates host immune response to post viral bacterial Pneumonia via IL-23/Th17 pathway.
- IRAK-M promotes alternative macrophage activation and fibroproliferation in bleomycin-induced Lung injury.
- Redundant and Cooperative Interactions between TLR5 and NLRC4 in Protective Lung Mucosal Immunity against Pseudomonas aeruginosa.
- Flagellin-induced expression of CXCL10 mediates direct fungal killing and recruitment of NK cells to the cornea in response to Candida albicans Infection.
- Epithelial-mesenchymal Transition Associated Secretory Phenotype Predicts Survival in Lung Cancer Patients.
- Extracellular histones are essential effectors of C5aR- and C5L2-mediated tissue damage and inflammation in acute Lung injury.
- Bronchoalveolar lavage neuregulin-1 is elevated in acute Lung injury and correlates with inflammation.
- Chest CT Features are Associated With Poorer Quality of Life in Acute Lung Injury Survivors*
- Genome-wide transcriptional analysis of differentially expressed genes in flagellin-pretreated mouse corneal epithelial cells in response to Pseudomonas aeruginosa: in...
- Danger-associated molecular patterns (DAMPs) in acute Lung injury.
- TLR9-Dependent IL-23/IL-17 Is Required for the Generation of Stachybotrys chartarum-Induced Hypersensitivity Pneumonitis.
- Fatty acids regulate stress resistance and virulence factor production for Listeria monocytogenes.
- Cathelicidin-related antimicrobial Peptide is required for effective Lung mucosal immunity in gram-negative bacterial pneumonia.
- The function of neutrophils in sepsis.
- TLR Signaling Prevents Hyperoxia-Induced Lung Injury by Protecting the Alveolar Epithelium from Oxidant-Mediated Death.
- Breaking the tolerance for tumor: Targeting negative regulators of TLR signaling.
- Role of TLRs in Brucella mediated murine DC activation in vitro and clearance of Pulmonary infection in vivo.
- TLR4-DEPENDENT GM-CSF PROTECTS AGAINST Lung INJURY IN GRAM-NEGATIVE BACTERIAL PNEUMONIA.
- Stachybotrys chartarum-Induced Hypersensitivity Pneumonitis Is TLR9 Dependent.
- Identifying Inhibitors of Epithelial-Mesenchymal Transition by Connectivity Map-Based Systems Approach.
- A randomized trial of recombinant human granulocyte-macrophage colony-stimulating factor for patients with acute Lung injury.
- The Emerging Field of Quantitative Blood Metabolomics for Biomarker Discovery in Critical Illnesses.
- Leptin in fibroproliferative acute respiratory distress Syndrome: not just a satiety factor.
- Toll like receptors in Diseases of the lung.
- Metabolic consequences of sepsis-induced acute Lung injury revealed by plasma ¹H-nuclear magnetic resonance quantitative metabolomics and computational ana...
- Peroxisome proliferator-activated receptor-gamma activation inhibits tumor metastasis by antagonizing Smad3-mediated epithelial-mesenchymal transition.
- PostInfluenza bacterial pneumonia: host defenses gone awry.
- TLR9 signaling is required for generation of the adaptive immune protection in Cryptococcus neoformans-infected Lungs.
- Flagellin stimulates protective Lung mucosal immunity: role of cathelicidin-related antimicrobial peptide.
- Nrf2 and PPAR{gamma}: PPARtnering against oxidant-induced Lung injury.
- A role for IL-1 receptor-associated kinase-M in prostaglandin E2-induced immunosuppression post-bone marrow transplantation.
- Critical role of IL-1 receptor-associated kinase-M in regulating chemokine-dependent deleterious inflammation in murine Influenza pneumonia.
- Curcumin inhibits fibrosis-related effects in IPF fibroblasts and in mice following bleomycin-induced Lung injury.
- Effects of sepsis on neutrophil chemotaxis.
- Therapeutic efficacy of Cintredekin Besudotox (IL13-PE38QQR) in murine Lung fibrosis is unaffected by immunity to Pseudomonas aeruginosa exotoxin A.
- Cooperative interactions between TLR4 and TLR9 regulate interleukin 23 and 17 production in a murine model of gram negative bacterial Pneumonia.
- IRAK-M regulates chromatin remodeling in Lung macrophages during experimental sepsis.
- Toll-like receptor 9 activation is a key mechanism for the maintenance of Chronic lung inflammation.
- Effect of IL-10 on neutrophil recruitment and survival after Pseudomonas aeruginosa challenge.
- Toll-like receptor 9 modulates immune responses to Aspergillus fumigatus conidia in immunodeficient and allergic mice.
- TLR9 regulates the mycobacteria-elicited Pulmonary granulomatous immune response in mice through DC-derived Notch ligand delta-like 4.
- Temporal quantitative proteomics by iTRAQ 2D-LC-MS/MS and corresponding mRNA expression analysis identify post-transcriptional modulation of actin-cytoskeleton regulat...
- Sepsis-induced inhibition of neutrophil chemotaxis is mediated by activation of peroxisome proliferator-activated receptor-{gamma}.
- Lung resident mesenchymal stem cells isolated from human Lung allografts inhibit T cell proliferation via a soluble mediator.
- Toll-like receptor 9 regulates the Lung macrophage phenotype and host immunity in murine pneumonia caused by Legionella pneumophila.
- PPAR-gamma agonists inhibit profibrotic phenotypes in human lung fibroblasts and bleomycin-induced Pulmonary fibrosis.
- Cyclic di-GMP stimulates protective innate immunity in bacterial Pneumonia.
- TLR9 is required for protective innate immunity in Gram-negative bacterial Pneumonia: role of dendritic cells.
- PPARs in alveolar macrophage biology.
- PPARs in Lung Biology and Disease.
- Sepsis-induced suppression of Lung innate immunity is mediated by IRAK-M.
- Role of granulocyte macrophage colony-stimulating factor during gram-negative Lung infection with Pseudomonas aeruginosa.
- Differential protein expression profiling by iTRAQ-2DLC-MS/MS of Lung cancer cells undergoing epithelial-mesenchymal transition reveals a migratory/invasive phenotype.
- Constitutive activation of prosurvival signaling in alveolar mesenchymal cells isolated from patients with nonresolving acute respiratory distress Syndrome.
- The lupus-susceptibility locus, Sle3, mediates enhanced resistance to bacterial Infections.
- Interferon-inducible protein 10, but not monokine induced by gamma interferon, promotes protective type 1 immunity in murine Klebsiella Pneumoniae Pneumonia.
- Defective innate antibacterial host responses during murine Klebsiella Pneumoniae bacteremia: tumor necrosis factor (TNF) receptor 1 deficiency versus therapy with ant...
- Legionella pneumophila evades gamma interferon-mediated growth suppression through interleukin-10 induction in bone marrow-derived macrophages.
- PPAR-gamma activation inhibits angiogenesis by blocking ELR+CXC chemokine production in non-small cell Lung cancer.
- The systemic response to Lung infection.
- Modulation of prosurvival signaling in fibroblasts by a protein kinase inhibitor protects against fibrotic tissue injury.
- IP-10 mediates selective mononuclear cell accumulation and activation in response to intraPulmonary transgenic expression and during adenovirus-induced Pulmonary infla...
- Peroxisome proliferator-activated receptor-{gamma} as a regulator of Lung inflammation and repair.
- Azithromycin blocks neutrophil recruitment in Pseudomonas endobronchial Infection.
- CpG oligodeoxynucleotides stimulate protective innate immunity against Pulmonary Klebsiella infection.
- STAT4 is a critical mediator of early innate immune responses against Pulmonary Klebsiella infection.
- Deactivation of murine alveolar macrophages by peroxisome proliferator-activated receptor-gamma ligands.
- Immunomodulation for the prevention and treatment of Lung infections.
- Defective phagocytosis and clearance of Pseudomonas aeruginosa in the Lung following bone marrow transplantation.
- The Pseudomonas aeruginosa autoinducer N-3-oxododecanoyl homoserine lactone accelerates apoptosis in macrophages and neutrophils.
- Murine complement interactions with Pseudomonas aeruginosa and their consequences during Pneumonia.
- Anti-tumor necrosis factor-alpha therapy during murine Klebsiella Pneumoniae bacteremia: increased mortality in the absence of liver injury.
- Increased mortality and dysregulated cytokine production in tumor necrosis factor receptor 1-deficient mice following Systemic Klebsiella pneumoniae infection.
- Sublethal hyperoxia impairs Pulmonary innate immunity.
- Hyperoxia mediates acute Lung injury and increased lethality in murine Legionella pneumonia: the role of apoptosis.
- IntraPulmonary expression of macrophage inflammatory protein 1alpha (CCL3) induces neutrophil and NK cell accumulation and stimulates innate immunity in murine bacteri...
- Divergent role of gamma interferon in a murine model of Pulmonary versus systemic Klebsiella pneumoniae infection.
- Roles of interleukin-6 and macrophage inflammatory protein-2 in pneumolysin-induced Lung inflammation in mice.
- Use of Animal Models in the Study of Inflammatory Mediators of Pneumonia.
- Cecal Ligation and Puncture Results in Long-Term Central Nervous System Myeloid Inflammation.
- Linezolid has unique immunomodulatory effects in post-Influenza community acquired MRSA pneumonia.
- Interferon Regulatory Factor-1 in Flagellin-Induced Reprogramming: Potential Protective Role of CXCL10 in Cornea Innate Defense Against Pseudomonas aeruginosa Infection.
Schools
Wayne State University School Of Medicine
University Of Minnesota
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asbestosis
- Asthma
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
Education
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University of Michigan Hospitals and Health Centers -
Wayne State University School of Medicine
Hospital
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University of Michigan Hospitals and Health Centers
Drug Facts
| NPI NUMBER | 1922180454 | |
| NPPES Provider LastName | STANDIFORD | |
| NPPES Provider FirstName | THEODORE | |
| NPPES Provider ZIPCode | 481095360 | |
| NPPES Provider State | MI | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 257.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1922180454 |
| Last Name Of The Provider | STANDIFORD |
| First Name Of The Provider | THEODORE |
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