Physician
Pediatric Emergency Medicine
Dr. Virginia M. Sanders, MD
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Virginia Sanders practices Pediatric Emergency Medicine. She graduated from Medical University of South Carolina. Virginia Sanders has written publications. She has 73 publications published with PubMed. The lastest publication is: 'Nonradioactive Northern blotting with biotinylated and digoxigenin-labeled RNA probes.'
Publications
- SOD1(G93A) transgenic mouse CD4(+) T cells mediate neuroprotection after facial nerve axotomy when removed from a suppressive peripheral microenvironment.
- Alloprimed CD8(+) T Cells Regulate Alloantibody and Eliminate Alloprimed B Cells Through Perforin- and FasL-Dependent Mechanisms.
- Axotomy-induced target disconnection promotes an additional death mechanism involved in motoneuron degeneration in ALS transgenic mice.
- Neuroendocrine regulation of inflammation.
- Adrenergic Regulation of IgE Involves Modulation of CD23 and ADAM10 Expression on Exosomes.
- Prohibitins and the cytoplasmic domain of CD86 cooperate to mediate CD86 signaling in B lymphocytes.
- Immune cell-mediated neuroprotection is independent of estrogen action through estrogen receptor-alpha.
- The beta2-adrenergic receptor on T and B lymphocytes: Do we understand it yet?
- Differential gene expression in the axotomized facial motor nucleus of pre-symptomatic SOD1 mice.
- Epigenetic regulation of beta2-adrenergic receptor expression in T(H)1 and T(H)2 cells.
- CD8+ T cells negatively regulate IL-4-dependent, IgG1-dominant posttransplant alloantibody production.
- Use of laser microdissection in the investigation of facial motoneuron and neuropil molecular phenotypes after peripheral axotomy.
- IL-10 within the CNS is necessary for CD4(+) T cells to mediate neuroprotection.
- Multifunctional microbubbles for image-guided antivascular endothelial growth factor therapy.
- Functional recovery and facial motoneuron survival are influenced by immunodeficiency in crush-axotomized mice.
- Effects of facial nerve axotomy on Th2-associated and Th1-associated chemokine mRNA expression in the facial motor nucleus of wild-type and presymptomatic SOD1 mice.
- Hematopoietic protein tyrosine phosphatase mediates beta2-adrenergic receptor-induced regulation of p38 mitogen-activated protein kinase in B lymphocytes.
- Differential actions of pituitary adenylyl cyclase-activating polypeptide and interferon gamma on Th2- and Th1-associated chemokine expression in cultured murine micro...
- Phenotype of CD4+ T cell subsets that develop following mouse facial nerve axotomy.
- Impaired antibody synthesis after spinal cord injury is level dependent and is due to sympathetic nervous system dysregulation.
- Autonomic innervation and regulation of the immune system (1987-2007).
- CD86 regulates IgG1 production via a CD19-dependent mechanism.
- Immune-mediated neuroprotection of axotomized mouse facial motoneurons is dependent on the IL-4/STAT6 signaling pathway in CD4(+) T cells.
- The level of IgE produced by a B cell is regulated by norepinephrine in a p38 MAPK- and CD23-dependent manner.
- Epigenetic regulation of Th1 and Th2 cell development.
- It takes nerve to tell T and B cells what to do.
- CD86 stimulation on a B cell activates the phosphatidylinositol 3-kinase/Akt and phospholipase C gamma 2/protein kinase C alpha beta signaling pathways.
- Role of immunity in recovery from a peripheral nerve injury.
- Interdisciplinary research: noradrenergic regulation of adaptive immunity.
- Brain-derived neurotrophic factor supports facial motoneuron survival after facial nerve transection in immunodeficient mice.
- Role of the immune system in the maintenance of mouse facial motoneuron viability after nerve injury.
- CD86 and beta2-adrenergic receptor signaling pathways, respectively, increase Oct-2 and OCA-B Expression and binding to the 3'-IgH enhancer in B cells.
- CD4-positive T cell-mediated neuroprotection requires dual compartment antigen presentation.
- Natural killer cells do not mediate facial motoneuron survival after facial nerve transection.
- CD4+ T, but not CD8+ or B, lymphocytes mediate facial motoneuron survival after facial nerve transection.
- Selective regulation of mature IgG1 transcription by CD86 and beta 2-adrenergic receptor stimulation.
- Adaptive immunity in mice lacking the beta(2)-adrenergic receptor.
- Functional recovery after facial nerve crush is delayed in severe combined immunodeficient mice.
- Norepinephrine, the beta-adrenergic receptor, and immunity.
- B cell receptor- and beta 2-adrenergic receptor-induced regulation of B7-2 (CD86) expression in B cells.
- Sympathetic nervous system interaction with the immune system.
- Delayed functional recovery in presymptomatic mSOD1(G93A) mice following facial nerve crush axotomy.
- Discovery of a new boron-containing antifungal agent, 5-fluoro-1,3-dihydro-1-hydroxy-2,1- benzoxaborole (AN2690), for the potential treatment of onychomycosis.
- Nonradioactive Northern blotting with biotinylated and digoxigenin-labeled RNA probes.
- Early detection of fractures with low-dose digital X-ray images in a pediatric trauma unit.
- Production of digoxigenin-labelled RNA probes and the detection of cytokine mRNA in rat spleen and brain by in situ hybridization.
- A pilot study evaluating the "STATSCAN" digital X-ray machine in paediatric polytrauma.
- Digital radiographic measurement of the atlantodental interval in children.
- CD4(+) T cell-mediated facial motoneuron survival after injury: Distribution pattern of cell death and rescue throughout the extent of the facial motor nucleus.
- Digital radiographic measurement of the main bronchi: a pilot study.
- Norepinephrine and beta 2-adrenergic receptor stimulation regulate CD4+ T and B lymphocyte function in vitro and in vivo.
- IFN-gamma production by Th1 cells generated from naive CD4+ T cells exposed to norepinephrine.
- Cytokine production by naive and primary effector CD4+ T cells exposed to norepinephrine.
- Norepinephrine: a messenger from the brain to the immune system.
- Kinetics of facial motoneuron loss following facial nerve transection in severe combined immunodeficient mice.
- Activation of antigen-specific CD4+ Th2 cells and B cells in vivo increases norepinephrine release in the spleen and bone marrow.
- Stimulation of the B cell receptor, CD86 (B7-2), and the beta 2-adrenergic receptor intrinsically modulates the level of IgG1 and IgE produced per B cell.
- Exacerbation of facial motoneuron loss after facial nerve transection in severe combined immunodeficient (scid) mice.
- Suppression of antigen-specific Th2 cell-dependent IgM and IgG1 production following norepinephrine depletion in vivo.
- Activated T helper 1 and T helper 2 cells differentially express the beta-2-adrenergic receptor: a mechanism for selective modulation of T helper 1 cell cytokine produ...
- Differential expression of the beta2-adrenergic receptor by Th1 and Th2 clones: implications for cytokine production and B cell help.
- Ultralow concentrations of proenkephalin and [met5]-enkephalin differentially affect IgM and IgG production by B cells.
- The role of norepinephrine and beta-2-adrenergic receptor stimulation in the modulation of Th1, Th2, and B lymphocyte function.
- Tissue specific effects of the beta 2-adrenergic agonist salbutamol on LPS-induced IFN-gamma, IL-10 and TGF-beta responses in vivo.
- CD86 and beta2-adrenergic receptor stimulation regulate B-cell activity cooperatively.
- CXCR3-/- mice mount an efficient Th1 response but fail to control Leishmania major infection.
- CD4+CD25+ regulatory T cells and CD1-restricted NKT cells do not mediate facial motoneuron survival after axotomy.
- Spinal cord injury triggers systemic autoimmunity: evidence for chronic B lymphocyte activation and lupus-like autoantibody synthesis.
- Stress hormones collaborate to induce lymphocyte apoptosis after high level spinal cord injury.
- Effects of facial nerve axotomy on Th2- and Th1-associated chemokine expression in the facial motor nucleus of wild-type and presymptomatic mSOD1 mice.
- Toll-like receptor 2 and facial motoneuron survival after facial nerve axotomy.
- Exacerbation of facial motoneuron loss after facial nerve axotomy in CCR3-deficient mice.
Schools
University of Tennessee Memphis College of Medicine amp Surgery
Doctors Specialties
- Anesthesiology
- Pediatrics
- Pediatric Emergency Medicine
Accepted Insurances
Education
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University of Tennessee -
Medical University of South Carolina -
University of Tennessee College of Medicine
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