Physician

Internal Medicine

Jaroslav G. Vostal

Jaroslav Vostal practices Internal Medicine. Jaroslav Vostal is a published doctor as well. He has 50 publications published. The latest was: Increased expression of phosphatidylinositol-specific phospholipase C resistant prion proteins on the surface of activated platelets.

Publications

Expression of the cellular prion protein affects posttransfusion recovery and survival of red blood cells in mice.
Inactivation of bacteria via photosensitization of vitamin K3 by UV-A light.
Human platelets pathogen reduced with riboflavin and ultraviolet light do not cause acute lung injury in a two-event SCID mouse model.
Activation of platelet protein kinase C by ultraviolet light B mediates platelet transfusion-related acute lung injury in a two-event animal model.
Underestimation of the expression of cellular prion protein on human red blood cells.
Rejuvenation improves roller pump-induced physical stress resistance of fresh and stored red blood cells.
Ultraviolet B light-exposed human platelets mediate acute lung injury in a two-event mouse model of transfusion.
Current and future cellular transfusion products.
Reduced erythroid cell and erythropoietin production in response to acute anemia in prion protein-deficient (Prnp-/-) mice.
Divergent expression of cellular prion protein on blood cells of human and nonhuman primates.
In vivo recovery of human platelets in severe combined immunodeficient mice as a measure of platelet damage.
Efficacy evaluation of current and future platelet transfusion products.
Endogenous osteonectin/SPARC/BM-40 expression inhibits MDA-MB-231 breast cancer cell metastasis.
FDA approach to evaluation of pathogen reduction technology.
Bacterial contamination of blood components: risks, strategies, and regulation: joint ASH and AABB educational session in transfusion medicine.
Activated platelets of patients with paroxysmal nocturnal hemoglobinuria express cellular prion protein.
Scrapie infectivity in hamster blood is not associated with platelets.
Cellular prion protein is expressed on endothelial cells and is released during apoptosis on membrane microparticles found in human plasma.
Toxicity of a first-generation adenoviral vector in rhesus macaques.
Adenoviral vectors do not induce, inhibit, or potentiate human platelet aggregation.
In vitro and in vivo characterization of ultraviolet light C-irradiated human platelets in a 2 event mouse model of transfusion.
A multistate cluster of red blood cell transfusion reactions associated with use of a leucocyte reduction filter.
Expression of cellular prion protein on platelets from patients with gray platelet or Hermansky-Pudlak syndrome and the protein''s association with alpha-granules.
Elevated circulating endothelial membrane microparticles in paroxysmal nocturnal haemoglobinuria.
Human cellular prion protein interacts directly with clusterin protein.
Cold temperatures reduce the sensitivity of stored platelets to disaggregating agents.
Release of annexin V-binding membrane microparticles from cultured human umbilical vein endothelial cells after treatment with camptothecin.
CD34+ cells from paroxysmal nocturnal hemoglobinuria (PNH) patients are deficient in surface expression of cellular prion protein (PrPc).
Expression of cellular prion protein on blood cells: potential functions in cell physiology and pathophysiology of transmissible spongiform encephalopathy diseases.
Biological action of nitric oxide donor compounds on platelets from patients with sickle cell disease.
Transmission of BSE by blood transfusion.
Different levels of prion protein (PrPc) expression on hamster, mouse and human blood cells.
Summary of a workshop on in vivo efficacy of transfused platelet components and platelet substitutes.
Restoration of in vitro responses in platelets stored in plasma.
Increased expression of phosphatidylinositol-specific phospholipase C resistant prion proteins on the surface of activated platelets.
Hemoglobin A0 and alpha-crosslinked hemoglobin (alpha-DBBF) potentiate agonist-induced platelet aggregation through the platelet thromboxane receptor.
Liquid cold storage of platelets: a revitalized possible alternative for limiting bacterial contamination of platelet products.
Peroxynitrite-induced tyrosine nitration and phosphorylation in human platelets.
Endogenous ADP prevents PGE1-induced tyrosine dephosphorylation of focal adhesion kinase in thrombin-activated platelets.
Thapsigargin-induced calcium influx in the absence of detectable tyrosine phosphorylation in human platelets.
Econazole inhibits thapsigargin-induced platelet calcium influx by mechanisms other than cytochrome P-450 inhibition.
Vinculin is a major platelet protein that undergoes Ca(2+)-dependent tyrosine phosphorylation.
Novel peptides derived from a region of local homology between uteroglobin and lipocortin-1 inhibit platelet aggregation and secretion.
Anion channel blockers cause apparent inhibition of exocytosis by reacting with agonist or secretory product, not with cell.
Western blot identification of platelet proteins that bind normal serum immunoglobulins. Characteristics of a 95-Kd reactive protein.
Prothrombin plasma clearance is not mediated by hepatic asialoglycoprotein receptors.
Cytosolic and stored calcium antagonistically control tyrosine phosphorylation of specific platelet proteins.
Intravenous administration of replication-incompetent adenovirus to rhesus monkeys induces thrombocytopenia by increasing in vivo platelet clearance.
Serial oxygen equilibrium and kinetic measurements during RBC storage.

Schools

Wayne State University

Doctors Specialties

Clinical Pharmacology
Internal Medicine

Accepted Insurances

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